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Titlebook: Superantigens; Bernhard Fleischer,Hans Olov Sjögren Book 1991 The Editor(s) (if applicable) and The Author(s), under exclusive license to

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sirable robustness and performance properties across a large range of operating conditions. The approach is based on an inner/outer loop control structure. Control redistribution or the allocation of multiple/redundant control effectors is governed by a control selector. The control selector is base
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The Anti-CD3-Induced Syndrome: A Consequence of Massive In Vivo Cell Activation, pneumopathy (. et al. 1989), infectious bacterial endotoxin (.-. 1968; . et al. 1975), tuberculosis (. et al. 1989), malaria (. et al. 1990), leishmaniosis (. et al. 1989), or immunological conditions such as graft vs host disease (. et al. 1989b).
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T Cell Recognition of Superantigens, most polymorphic domains of the MHC molecules (α1 and 2 for MHC class I molecules, and α1 and β1 for MHC class II molecules) (. et al. 1987a, b; Brown et al. 1988). The TCR recognizes this MHC/peptide complex (. and . 1988).
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,Molecular Genetics of Pyrogenic Exotoxin “Superantigens” of Group A Streptococci and ,, elaboration by the microbe of toxins which induce disease manifestations. In this example the pathogen must only be able to survive and multiply within the host for a sufficient period of time to make the toxin. Toxin-host cell receptor interactions then determine the extent of host tissue damage.
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T Cell Recognition of Superantigens,ir ligand specificity: While the Ag receptors of B lymphocytes (immunoglobulins, Ig) bind to soluble Ag, the T cell Ag receptors (TCR) recognize Ag presented by cells in conjunction with products of the polymorphic major histocompatibility complex class I and class II genes (MHC class I/II) (. and .
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