Titlebook: Subcellular Basis of Contractile Failure; Proceedings of the S Borivoj Korecky,Naranjan S. Dhalla Conference proceedings 1990 Kluwer Academ

清洗

Role for Sodium Channels and Intracellular Sodium in Regulation of the Cardiac Force-Frequency Relatmulation frequency leads to an increase in twitch tension amplitude that requires a few minutes to reach a steady-state. Lowering of the stimulation frequency results in an opposite effect with a gradual decrease in twitch tension amplitude. Therefore, over a wide range of stimulation frequencies th

伪造

jarring

Hormonal and Non-Hormonal Regulation of Ca2+ Current and Adenylate Cyclase in Cardiac Cellsaline and acetylcholine (ACh) is in part mediated by the effects of these neurotransmitters on calcium current, I. (1). β-adrenergic stimulation of I. is mediated by a guanine-nucleotide binding protein, G. (2), which triggers the activation of adenylate cyclase (AC) and in turn stimulates cAMP-depe

bifurcate

Detoxification

GEN

其他

Modifications in Sarcolemmal Regulation of Ca2+ with Agingne in peak diastolic filling rate with age without the impairment of systolic function (1,4). Many of the animal studies have employed rat model, in which the changes in myocardial function and metabolism occur as early as during adult maturation (2,5,6) and progress through senescence (3). At about

Mri485

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使饥饿

Molecular and Subcellular Mechanisms of Thyroid Hormone Induced Cardiac Alterationsarly been long recognized (2). Since the cardiac effects of thyroid hormone excess and deficiency are prominent, considerable research has focused on the mechanism by which thyroid hormone induces these changes. A number of reviews have been published which summarize the clinical and experimental li