Titlebook: Studies on Respiratory Disorders; Nirmal K. Ganguly,Surinder K. Jindal,Ruby Pawankar Book 2014 Springer Science+Business Media New York 20

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Role of Exhaled Biomarkers, Volatiles, and Breath Condensate, noninvasive. This approach is further strengthened by an increasing evidence of the correlation between exhaled biomarker patterns and the occurrence of certain diseases and disorders. Exhaled biomarkers include both volatile and nonvolatile molecules. Exhaled gases, such as nitric oxide and carbon

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,Pulmonary Infections—Oxidant Injury and Role of Antioxidants,t role in the injurious and inflammatory responses in lung diseases. The normal system is provided with a defensive control in the form of protective antioxidant machinery. This comprises of enzymatic and non-enzymatic molecules that control the level of oxidative stress generated by reactive specie

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Oxidative Stress in Tuberculosis,g a successful intracellular pathogen, has evolved a number of antioxidant strategies to avoid killing by ROS produced by host cells. At the same time, excess of these ROS can result in necrosis of granulomatous lesions causing dissemination of bacilli from the granulomas. A number of antioxidant me

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Air Pollution and Oxidative Stress in Allergic Airway Diseases,children in both the developed and developing, allergic diseases comprise a global public health issue. The increase in prevalence of allergic diseases contributes to reduced quality of life of the patients and increased socioeconomic costs. Several factors contribute to this rise in prevalence of a

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Oxidative Stress in Sarcoidosis,of oxidative stress including lipid peroxidation products (like malondialdehyde, 8-isoprostane) and oxygen-free radicals (like hydrogen peroxide, superoxide) have been found in the serum and respiratory samples of sarcoidosis patients. Some of these markers correlate with disease activity. Enzymatic

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Asbestos Fibers: Mechanisms of Injury,d configuration (serpiginous vs. needle-shaped), as well as the pulmonary response of clearance, breakdown, or incorporation into the pulmonary interstitium all play a role in determining whether the cellular processes of acute and chronic inflammation, fibrosis, and genotoxicity occur in the lung.