Titlebook: Stroke Genetics; Pankaj Sharma,James F. Meschia Book 20172nd edition The Editor(s) (if applicable) and The Author(s), under exclusive lice

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Stroke-Like Episodes in Mitochondrial Myopathy, Encephalopathy, Lactic Acidosis, and Stroke-Like Epes [1]. The factors that mediate the pathogenesis and mechanism of MELAS syndrome remain elusive, as have effective therapies. In this review, we discuss the current basis of understanding and theories relating to the common phenotypic features of MELAS, in particular the pathognomonic "stroke-like

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White Matter Disease, functional integrity of the WM is bound to present with diverse neurological manifestations. The range of neurological dysfunction varies greatly from relentless failure to thrive in a spectrum of pediatric leukodystrophies to subtle cognitive decline in the elderly with leukoaraiosis. Pathophysiol

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Genetics of Cervical Artery Dissection,d adults. Its risk factors and underlying mechanisms are poorly understood. Seldom, CeAD can be caused by a monogenic connective tissue disease, mainly vascular Ehlers-Danlos syndrome. In the absence of a known monogenic connective tissue disorder, a small proportion of CeAD patients (<3%) have a fa

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Cerebral Venous Thrombosis: Genetic Aspects,g diagnosed with higher frequency due to increased awareness and ready access to MR. Multiple conditions have been reported in association with CVT. Risk factors and associated conditions for CVT can be categorized as permanent or transient. The most frequent permanent risk factors are genetic proth

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Genetic Research and the Law,e research be conducted in a way that protects the privacy of the participants. The main way to achieve that goal is to take steps to ensure that no one discloses confidential genetic information. That task is made more difficult by the fact that researchers are turning to electronic databases to ke