Titlebook: Stroke Genetics; Pankaj Sharma,James F. Meschia Book 20172nd edition The Editor(s) (if applicable) and The Author(s), under exclusive lice

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Sickle Cell Disease,in SCD. All of these contributes to create significant phenotypic variability both in the “parent” SCD and the associated complication of stroke and cerebrovascular disease. More research is required to enable us understand the biological complexity of SCD in general and of the associated complication of stroke in particular.

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Book 20172nd editionstudies, and also monogenic disorders. Finally, the legal and ethical complexities in dealing with these issues are discussed..Stroke Genetics. is a valuable resource for neurologists, stroke physicians, hypertension specialists, internists, clinical pharmacologists and those in training, as well as researchers in the field of disease genetics..

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tion.Includes new chapters on nonCaucasian stroke, advanced This revised, expanded second edition updates the reader on this fast moving field as well providing an overall understanding of the genetics of complex diseases by using stroke as a paradigm. The reader will gain a comprehensive understand

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Familial Occurrence and Heritability of Stroke, risk factors, or stroke subtype [2, 3]. Focused investigations within these subgroups using modern genotyping techniques (phenotyping ‘splitting’) can increase the yield in identifying gene variants associated with stroke [4, 5]. As a predisposing risk factor, family history can be used clinically

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Other Monogenetic Stroke Disorders,inant etiologic mechanisms, including large arterial diseases, small vessel diseases, hematological diseases, mitochondrial diseases, and connective tissue disorders. The disorders are presented in the same order in both tables. For completeness and ease of reference, we have also included several m

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White Matter Disease,been linked to risk of ischemic and hemorrhagic strokes. This subtype of WM disease, also known as WM lesions (WMLs), WM hyperintensity (WMH), or leukoaraiosis, has been described in the context of epidemiological studies of aging adults with subtle, but progressive, signs of neurological dysfunctio

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Genetics of Cervical Artery Dissection, association studies have been published, on relatively small samples, which did not reveal any robust associations. Recently, the first genome-wide association study (GWAS) of CeAD was published, resulting from a large international effort gathering samples from over 2000 CeAD patients. This GWAS i

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