Titlebook: Stroke; Emergency Management Thorsten Steiner,Werner Hacke,Daniel F. Hanley Book 1998 Springer-Verlag Berlin Heidelberg 1998 Hirnschlag.Hir

jabber

Cytoprotective Drugs After Subarachnoid Hemorrhage, of free radicals. The systems that produce reperfusion injury can develop free radicals. Nitric oxide itself can function as a free radical and can increase the production of others. Lipid peroxidation can result in more extracellular glutamate release.

佛刊

Intracranial Pressure and Circulatory Management,ding intravascular volume expansion to maintain cerebral perfusion and oxygen delivery to areas of ischemic tissue, with the goal of preventing reversible ischemia from progressing to irreversible infarction.

精致

ENNUI

Operative and Interventional Neuroradiology,essel preparation. Factors that contribute to a good outcome following subarachnoid hemorrhage (SAH) include successful repair of the primary bleeding site, early clot removal, prevention of vasospasm, and prevention of rebleeding. Treatment approaches include both surgical and endovascular modalities.

干涉

Aerophagia

Improving Early Referral and Emergency Care in Acute Stroke, require earlier stroke detection by the public, faster transportation of the stroke patient to the hospital, faster evaluation by emergency room staff, and prompt administration of therapeutic agents. Improving stroke referral and treatment systems is key to achieving maximum benefits from these ne

不透明性

ineluctable

高射炮

Autonomic Changes in Acute Cerebrovascular Disorders,nd ischemic stroke. The common basis for these problems is maximal activation of the sympathetic pathway, with parasympathetic coactivation. A major main clinical symptom is electrocardiograph (ECG) change that may include a long QT interval, torsades des points, ventricular arrhythmias (Fig. 1), an

闪光东本

Intracranial Pressure and Circulatory Management,gency treatment of acute stroke. The key elements are to maintain cerebral perfusion pressure (CPP) above 70 mmHg and to use hemodynamic therapy including intravascular volume expansion to maintain cerebral perfusion and oxygen delivery to areas of ischemic tissue, with the goal of preventing revers