Titlebook: Spinal Disorders in Growth and Aging; Hideaki E. Takahashi Conference proceedings 1995 Springer Japan 1995 Spinal disorders.biomechanics.g

说笑

Cotrel-Dubousset Instrumentation in Trauma, Metastatic Tumor, and Degenerative Spine Diseaseility. On the other hand we had been anxious about the burden of the operative procedure which would increase operating time, increase blood loss, and cause more complications. The purpose of this report is to assess the utility of C-D-I in various pathologies, except scoliosis, from the viewpoint of operating stress and complication.

reception

积极词汇

Stress Analysis of the Lumbar Spine Using the Finite Element Modeles including instrumentation are also important. The finite element method (FEM) is a simulation technique that could provide a significant advantage to achieve these purposes [1–3]. Two- and three-dimensional FEM models were employed to simulate and evaluate the behavior of a normal lumbar spine before and after surgery.

万花筒

Microaneurysm

Takahiko Nakamura,Takao Homma,Seiji Uchiyama,Hideaki E. Takahashi

sperse

Sakae Sato,Kunio Ibaraki,Hiroaki Takara,Fuminori Kanaya,Satoshi Mori,Takahiko Nakamurastill requires intense effort on the development of the necessary technologies, but it is a realistic option to increase the energy output of different types of fusion reactors and to increase the cost efficiency. First of all we would like to give an overview on the current situation in energy prod

EVEN

擦试不掉

Yoshio Ota,Yoshihiko Oshima,Taro Nagashima,Masahiro Hayashi,Toshiaki Samotos ways of working through incomprehensible loss. The novel models a paradoxical approach to thinking the nuclear future—both mournful and defiant, paralyzed and unrelentingly active. Ozeki’s quantum poetics embodies an approach that confronts the unknowability of the future as well as the past.

abolish

Masahiro Hayashi,Yoshihiko Ohshima,Yoshio Ohta,Hiroshi Satoh,Noritoshi Hiramoto,Minoru Yokota,Tomokap to a week. This imaging platform was recently applied to study reversal of cardiac myocyte hypertrophy following withdrawal of the α-adrenergic agonist phenylephrine. Hypertrophy readily reversed at low but not high levels of α-adrenergic signaling, leading to identification of an intracellular po

Simulate