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Titlebook: Sphingolipid-Mediated Signal Transduction; Yusuf A. Hannun Book 1997 Springer-Verlag Berlin Heidelberg 1997 biology.cell.cell death.progra

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书目名称Sphingolipid-Mediated Signal Transduction
编辑Yusuf A. Hannun
视频video
丛书名称Molecular Biology Intelligence Unit
图书封面Titlebook: Sphingolipid-Mediated Signal Transduction;  Yusuf A. Hannun Book 1997 Springer-Verlag Berlin Heidelberg 1997 biology.cell.cell death.progra
描述Lipid-derived molecules are increasingly appreciated as key regulators of cell responses and signal transduction. Sphingolipid-derived bioactive molecules are emerging as critical regulators of some of the most exciting and, until recently, poorly-understood areas of biology such as growth suppression, programmed cell death and tumor invasion. Written by leading investigators in these areas of study, this book presents a thorough, current and accessible review of how sphingolipids function to regulate these important activities.
出版日期Book 1997
关键词biology; cell; cell death; programmed cell death; signal transduction
版次1
doihttps://doi.org/10.1007/978-3-662-22425-0
isbn_softcover978-3-662-22427-4
isbn_ebook978-3-662-22425-0Series ISSN 1431-0414
issn_series 1431-0414
copyrightSpringer-Verlag Berlin Heidelberg 1997
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Ceramide: A Stress Response Mediator Involved in Growth Suppression, A number of functions were associated with the glycosphingolipids, including a role in cellular interaction, differentiation, and oncogenic transformation.. Additionally, complex glycosphingolipids were shown to serve as antigens important in the immune response, as receptors for viruses or bacteria, and as tumor markers..
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Ceramide, a Mediator of Cytosine Arabinoside Induced Apoptosis,tion these signaling pathways are attractive targets for the improvement of therapy regimens and the development of new drugs.. It is our aim to determine the signaling pathways in response to chemotherapeutic agents with the goal of developing more effective drugs and new combinations of existing drugs.
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Ceramide, Aging and Cellular Senescence,del argues that alterations of genetic control programs could lead to cellular senescence. There is evidence to support both hypotheses, yet the underlying mechanisms of cellular senescence are still not clear.
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Introduction and Overview,bicity, poor aqueous solubility, and self-assembly) are critical to the formation of membrane bilayers which, in turn, define the boundaries of the cells as well as intracellular compartments. The lipids of the plasma membrane (which comprise glycerolipids, sphingolipids, and cholesterol) provide fo
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