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Titlebook: Sex Steroids and the Cardiovascular System; Peter W. Ramwell,Gabor M. Rubanyi,Ekkehard Schilli Conference proceedings 1992 Springer-Verlag

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Vascular Non-genomic Effects of Estrogen,ught to passively diffuse into the cell and bind to their nuclear receptor protein. Each receptor is both ligand and cell specific, and binds to its respective steroid with high affinity (..: 0.1–1.0 n.). The ligand-receptor complex becomes an activated transcription factor, which binds gene regulat
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Effects of Estrogens and Progestins on Atherosclerosis in Primates,usal women than in men of similar age. This gender difference is paralleled by a difference in extent and severity of coronary artery atherosclerosis (McGill and Stern 1979). It is widely believed that ovarian estrogen is responsible for this relative sparing of the coronary arteries; however, it re
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A Review of the Epidemiology of Postmenopausal Estrogens and the Risk of Coronary Heart Disease,al women, but rise sharply with age. Moreover, the ratio of rates between men and women grows smaller with increasing age (US Department of Health and Human Services 1988). This observation led to speculation that functioning ovaries in premenopausal women were protective. Although the risk of coron
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Vascular Non-genomic Effects of Estrogen,espective steroid with high affinity (..: 0.1–1.0 n.). The ligand-receptor complex becomes an activated transcription factor, which binds gene regulatory elements on DNA to enhance transcription of several target genes. Protein synthesis and processing follows (Moudgil 1987).
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0947-6075 ophysiological regulation of the cardiovascular system. The hor­ monal profile appears to put males at a disadvantage biologically in terms of cardiovascular diseases. The incidence of hypertension is higher in men than in pre-monopausal women indicating an influence of gender on the hypertensive di
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978-3-662-02766-0Springer-Verlag Berlin Heidelberg 1992
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Sex Steroids and the Cardiovascular System978-3-662-02764-6Series ISSN 0947-6075
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