Titlebook: Septic Shock; European View Jean Louis Vincent (Assistant-Director),Lambertus Conference proceedings 1987Latest edition Springer-Verlag Be

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Coronary Hemodynamics and Myocardial Metabolism in Septic Shock,ic pattern of septic shock in humans is generally characterized by a high cardiac output and a low systemic vascular resistance [1, 2]. The hyperdynamic circulation is not likely due to peripheral arterio-venous shunts, since, in skeletal muscle at least, capillary blood flow is increased, and varie

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Whole Body Energy Metabolism in the Hyperdynamic Phase of Sepsis,mal while glucose oxidation is somewhat inhibited and glucose uptake does not increase normally with insulin. Successful treatment often consists primarily in feeding the patient enough to sustain the hypermetabolic state as long as sepsis persists; and prognosis has improved markedly since total pa

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Microbiological Aspects of Septic Shock,to infection. Although bacteria are clearly involved in this process, interpretation of their role has changed in recent years, and it is now generally accepted that host defence and environmental factors also exert a major influence on the development of the syndrome. This chapter presents a clinic

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The Possible Roles of Lipoxygenase Products of Arachidonic Acid Metabolism and of Platelet Activatit is as yet too early to decide on their importance relative to other more established mediators. This brief review attempts to summarize the limited experimental data available on their actions and possible role in shock. Hopefully, it will provide pointers for more clearly defining their importance in tissue low perfusion states.

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Microbiological Aspects of Septic Shock,al appraisal of the microbiological aspects of septic shock in the context of general intensive care, with special reference to patients suffering from surgical complications or major trauma. No attempt is made to discuss laboratory aspects.

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The Role of Proteolytic Enzyme Systems with Particular Emphasis on the Plasma Kallikrein-Kinin Systtained from those who died of gram-negative septicemia had very low contents of high molecular weight kininogen [7]. In the patients surviving gram-negative septicemia, the blood kininogen levels, after an initial lowering, rose towards normal values.