Titlebook: Reviews of Physiology, Biochemistry and Pharmacology 131; Special Issue on Mem Book 1997 The Editor(s) (if applicable) and The Author(s),

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M. Ziegler,D. Jorcke,M. Schweigerif the interaction with the interface consists in drawing graphic elements, text input may be considered for some purposes, such as annotation, labelling, or input of recognizable text. This work deals with the detection of textual patterns in a set of digital ink elements. The main idea is that tex

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S. L. Oei,J. Griesenbeck,M. Schweigerby two parallel bold lines, symbols(door, window, tile...), dimension line, extension line, and dimensions represent various numerical values and characters. This paper suggests a method for recognizing main wall which is a back-bone of apartment in an architectural drawing. In this thesis, the foll

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F. Laval,D. A. Wink,J. Lavalting index is sought. Various contents formats for journals, which have different features from those for general documents, are described. The principal elements that we want to represent are titles, authors, and pages for each paper. Thus, the three principal elements are modeled according to the

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Reviews of Physiology, Biochemistry and Pharmacologyhttp://image.papertrans.cn/r/image/829644.jpg

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https://doi.org/10.1007/3-540-61992-5ADP-ribose; DNA repair proteins; oxidative damage; biochemistry; metabolism; pharmacology; physiology; rese

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A discussion of mechanisms of NO genotoxicty: Implication of inhibition of DNA repair proteins,riments were done under very high concentrations of NO and RNOS and may have little or nothing to do with in vivo mechanisms. However, NO can affect DNA specific repair systems even in whole cells at lower NO and RNOS concentrations which might enhance the damage of another agent. Though NO might al

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A discussion of mechanisms of NO genotoxicty: Implication of inhibition of DNA repair proteins,ter DNA directly, the most likely involvement of its genotoxic action is through the increase in sensitivity to other mutagenic agents. From this discussion, it appears that the primary source of RNOS is from iNOS. Thus genotoxicity either by direct chemical alteration of DNA or interference with the repair system would be from an iNOS source.