Titlebook: Resistance to Photodynamic Therapy in Cancer; Valentina Rapozzi,Giulio Jori Book 2015 Springer International Publishing Switzerland 2015 P

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书目名称Resistance to Photodynamic Therapy in Cancer影响因子(影响力)




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Tumor Microenvironment as a Determinant of Photodynamic Therapy Resistancetion status within the tumor, and expression and/or activation of proteins that mediate tumor progression can contribute to the efficacy of, or resistance to, various therapeutic modalities. Photodynamic therapy (PDT) is no exception—the oxygenation status and molecular makeup of the tumor and its s

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Isolation and Initial Characterization of Resistant Cells to Photodynamic Therapypotential mechanisms implicated in such resistance. In this sense, we describe the methods published for the isolation and characterization of tumor cells resistant to PDT. We also propose other unpublished procedures that could be of interest for the study of cells resistant to PDT. Factors such as

Banister

GRP78-targeting Sensitizes Cancer Cells to Cytotoxic Effects of Photodynamic Therapyygen and reactive oxygen species. Intracellular proteins have been shown to undergo oxidation-related damage in response to PDT. A number of cytoprotective mechanisms have been demonstrated to relay on mechanisms associated with removal or re-folding of these proteins or leading to the induction of

缩影

Optimization of Photodynamic Therapy Response by Survivin Genetional cancer therapies are based on targeting rapidly growing cancerous cells to block growth or enhance cell death, thereby, restoring the balance between these processes. In many instances, malignancies that develop resistance to current treatment modalities, for example photodynamic therapy (PDT

Antarctic

Cellular Targets and Molecular Responses Associated with Photodynamic Therapyes associated with the use of PDT and we have observed increased expression of several prosurvival molecules that can modulate treatment efficacy. Specifically, angiogenic growth factors, inflammatory proteins, and anti-apoptotic molecules are often overexpressed following PDT-mediated oxidative str

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