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Titlebook: Renal Medicine; E. N. Wardle Book 1979 E. N. Wardle 1979 dialysis.glomerulonephritis.hemodialysis.kidney.renal failure.transplantation

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nar, dynamic and thermal boundary layer. Historical perspectives are included whenever they enrich the understanding of modern concepts. Many examples, chosen for their pedagogical relevance, are dealt with in exercises..The book is intended as a teaching tool for undergraduate students, wishing to
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Book 1979rom this side of the Atlantic, which provide a really sound foundation of renal physiology, biochemistry and immunology. For two decades "The Kidney" by Hugh de Wardener has given a splendid background in renal physiology but I doubt if any author has brought to this subject as wide an experience an
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Haemodialysis, electrolyte composition is close to that of normal plasma. Urea and low molecular weight toxic products (of less than 1200 daltons) diffuse from the blood into the dialysate, whilst glucose and bicarbonate or acetate move in the opposite direction.
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Clinical varieties and treatment of nephritis, failure is also rare, although, depending on fluid intake and the urine output, there may be evidence of fluid overload in the form of raised jugular venous pressure, a dilated heart and some pulmonary oedema.
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Kidney stones and congenital diseases of the kidney,lternatively to regulate the level in the cytosol by an energy-dependent uptake into the mitochondria. Phosphate moves likewise. Conversely, if calcium is taken up by mitochondria independently of phosphate, hydrogen ions are then secreted into the cytosol.
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The kidneys in health and disease: renal physiology and pathophysiology,a tuft of glomerular capillaries which invaginate into an epithelial sac known as Bowman’s capsule which joins with the actual renal tubule. Each of the one million nephrons then drains via coalescing collecting tubules into the renal pelvis, so that finally the urine enters the ureter en route to t
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The kidney and hypertension,f the matter, however, is that the juxtaglomerular apparatus (Figure 22) consists of stretch receptors in the wall of the afferent arteriole working in conjunction with the sodium-sensitive cells of the macula densa, so that either (÷)diminished renal blood flow or (÷) a decrease in the sodium being
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Clinical varieties and treatment of nephritis, proliferative and exudative nephritis. The infecting organism is usually a beta-haemolytic streptococcus of Griffith’s M protein type 12. One week after a sore throat, or even earlier in some cases, the patient starts to pass brown ‘smoky’ urine which is indicative of haematuria. Collection of the
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