Titlebook: Regulation of Leukocyte Function; Ralph Snyderman Book 1984 Springer Science+Business Media New York 1984 Insomnia.anatomy.antimicrobial.b

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Transductional Mechanisms of Chemoattractant Receptors on Leukocytes,ury ago and formed the basis of Metchnikoff’s phagocytic theory of host defense (Metchnikoff, 1968). While the chemotactic migration of leukocytes was noted during the nineteenth century, it was not until 1962 that quantification of leukocyte Chemotaxis . became possible. The modern era of Chemotaxi

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The Neutrophil N-Formyl Peptide Receptor: Dynamics of Ligand-Receptor Interactions and Their Relati process. Because these peptides can be prepared synthetically, it has been possible to correlate extensively the structure-function relationships of a variety of amino acid sequences in both stimulatory and inhibitory peptides. Moreover,.this diversity in available sequences has permitted the devel

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Mechanisms of Leukocyte Regulation by Complement-Derived Factors,ocyte migration (Ward ., 1965, 1967; Shin ., 1968; Snyderman ., 1968). Shortly thereafter assignments were made that identified anaphylatoxins as the humoral chemotactic factors in question (Jensen ., 1969; Hill and Ward, 1969; Ward 1967, 1969; Cochrane and Müller-Eberhard, 1968; Fernandez ., 1978).

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Mechanisms of Regulating the Respiratory Burst in Leukocytes,henomenon was first observed as increased oxygen uptake by the stimulated cells (Baldridge and Gerard, 1933; Sbarra and Karnovsky, 1959) and was correlated with the production of hydrogen peroxide (Iyer .., 1961). Concomitant with the alterations in respiration, enhanced glucose oxidation via the he

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Nonoxidative Antimicrobial Reactions of Leukocytes,ikoff discovered the role of . in defense against infection, much research has focused on the mediation of their antimicrobial activities (Metchnikoff, 1905). At first this work sought what may now be thought of as antibioticlike components of . with direct antimicrobial action. Research emphasis th

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Clinical Disorders of Leukocyte Functions,formed him that I was writing another review on clinical abnormalities of phagocyte function. Why write another such review, I was asked, as there are a number of these in the current literature (Gallin, 1981; Lohr and Snyderman, 1981; Hill, 1981a; Gallin ., 1980; Hill, 1982; Charette and Hill, 1980