Titlebook: Recent Advances in Immunology; Asuman Ü. Müftüoğlu,Nefise Barlas Book 1984 Plenum Press, New York 1984 antibody.antigen.genetics.immunolog

CORD

Role of Macrophages in T Cell Activation,ting cells. However, beside MØ, other cells which are not considered to belong to the MØ lineage are also involved in the induction of at least some immune processes. Thus, these ‘inducer’ cells are heterogeneous and the use of the term ‘accessory cells’ (AC) or ‘antigen presenting cells’ (APC) inst

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DUST

,A Cell-Free Product Secreted by Ly−2+ Cells can Induce a Molecule Required for Ly2 Suppressor Cell s of this circuit have been identified by the correlation of function with a unique profile of cell surface glycoproteins (differentiation antigens). The induction signal is delivered by an I-J. Ly1.2. cell to an Ly1.2. I-J. acceptor cell (1,2). The interaction between the Ly1 inducer cell and the L

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Frequency-Analysis of Precursors of Cytotoxic T Lymphocytes in Radiation Chimeras: Enumeration of Aderstood. From studies of the CTL-immune responsiveness in thymus- and bone marrow-grafted chimeric mice, it became apparent, that it is the thymus which is crucial not only for the maturation or T cells, but also for the specificity repertoire of the T cells (1,2). From these data it was suggested,

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树木中

Molecular Cloning of H-2 Class I Genes in the H-2b Haplotype, which contain 40–45 kilobasepairs (kbp) of mouse DNA per clone, were chosen since there are about 20–30 class I genes (see below) in the mouse genome and most of these genes map between the H-2K and TL loci on chromosome 17 (Figure 1); a region of some 1000 kbp. of DNA. Estimates of the number of c

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Why Study Complement Genetics?,there is so much genetic variation there to study. Table 1 shows how common genetic polymorphisms are among human complement components. The term “polymorphism” was coined by E.B. Ford (1) to denote an allelic system where the second most common allele occurs at a frequency too high to be maintained

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The Family of Proteins Having Internal Thiolester Bonds, (2) later confirmed this sensitivity of C4 to nitrogen nucleophiles, and in 1953 both a plasmatic inhibitor of serum and “Factor A” of the properdin pathway were shown to be inactivated by hydrazine (3,4). However, it was not until 1968 that the plasmatic inhibitor was identified as α.-macroglobuli

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bronchodilator