Titlebook: Obesity: Pathology and Therapy; Dean H. Lockwood (Vice President),Thomas C. Heffne Book 2000 Springer-Verlag Berlin Heidelberg 2000 CNS.Fe

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Serotonin Drugs and the Treatment of Obesityan 20 years ago. Early studies showed that increasing CNS 5-HT levels by using 5-HT precursors such as tryptophan and 5-hydroxytryptophan (5-HTP) produced a significant reduction in the food intake of laboratory animals. Increasing 5-HT activity by other mechanisms such as directly administering 5-H

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咯咯笑

Diet and Body Weight Reductionas been the main guideline to plan dietary regimens in the context of weightreducing programs for decades. In dietetic practice, low-calorie and very lowcalorie diets represent two popular dietary approaches that are based on this guideline and that are still promoted by many agencies and health pro

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招待

Central Nervous System Neuropeptides Involved in Obesity has led to and helped our understanding of the pathophysiology of this complex system. Opinion has therefore changed from consideration of obesity as a self-inflicted problem to a view that it is a complicated disease with an associated increase in morbidity and mortality. In this chapter we review

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3 Adrenergic Receptors as a Therapeutic Target for Obesityon and release from white adipose tissue, and oxidation of fatty acids in brown fat. The net effect of adipocyte .AR activation is the reduction of triglyceride stores in white fat with a concomitant increase in thermogenesis and thermogenic capacity of brown fat. Because of the potential significan

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FAST

The Molecular and Epidemiological Genetics of Obesityritability level for human obesity. In the first part of this chapter, we will review the recent findings on genes linked or associated with obesity phenotypes. In the second part, the genetic epidemiology studies will be summarized.

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