Titlebook: Obesity and Lipotoxicity; Ayse Basak Engin,Atilla Engin Book 20171st edition The Editor(s) (if applicable) and The Author(s), under exclus

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Microbiota and Lipotoxicity,c lipid accumulation in non-adipose tissues. Low grade chronic inflammation and insulin resistance are the hallmarks of the disorder, which further aggravate the condition. Gut microbiota constitutes an indispensible part of human superorganism’s energy harvesting apparatus. The dynamic composition

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Insulin Resistance, Obesity and Lipotoxicity,everal metabolic pathways, both in adipose tissue and peripheral organs, like liver, heart, pancreas and muscle. Lipotoxicity has roles in insulin resistance and pancreatic beta cell dysfunction. Increased circulating levels of lipids and the metabolic alterations in fatty acid utilization and intra

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Diet-Induced Obesity and the Mechanism of Leptin Resistance,ated fatty acid can induce leptin resistance by activating negative feedback pathways. Although, long form leptin receptor (Ob-Rb) initiates leptin signaling through more than seven different signal transduction pathways, excessive suppressor of cytokine signaling-3 (SOCS-3) activity is a potential

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Influence of Antioxidants on Leptin Metabolism and its Role in the Pathogenesis of Obesity,terestingly, , food preservatives like sodium sulfite and sodium benzoate and also natural colorant and spice compounds such as curcumin were found to decrease the release of leptin in murine 3T3-L1 adipocytes, after co-incubation with LPS, which was added to mimic the pro-inflammatory status in obe

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