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Titlebook: Obesity and Lipotoxicity; Ayse Basak Engin,Atilla Engin Book 20171st edition The Editor(s) (if applicable) and The Author(s), under exclus

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发表于 2025-3-21 17:30:37 | 显示全部楼层 |阅读模式
书目名称Obesity and Lipotoxicity
编辑Ayse Basak Engin,Atilla Engin
视频videohttp://file.papertrans.cn/701/700122/700122.mp4
概述A comprehensive overview of obesity and lipotoxicity.Discusses the mechanisms of obesity and possibilities for the development of new treatment strategies.Overview of the biochemical pathways leading
丛书名称Advances in Experimental Medicine and Biology
图书封面Titlebook: Obesity and Lipotoxicity; Ayse Basak Engin,Atilla Engin Book 20171st edition The Editor(s) (if applicable) and The Author(s), under exclus
描述Due to the resultant health consequences and considerable increase in prevalence, obesity has become a major worldwide health problem. “Obesity and Lipotoxicity” is a comprehensive review of the recent researches to provide a better understanding of the lipotoxicity-related mechanisms of obesity and the potential for the development of new treatment strategies. This book overviews the biochemical pathways leading to obesity-related metabolic disorders that occur subsequent to lipotoxicity. Chapters examine the deleterious effects of nutrient excess at molecular level including the cellular and molecular aspects of breast cancer, resistance to leptin, insulin, adiponectin, and interconnection between the circadian clock and metabolic pathways during high-fat feeding. “Lipotoxicity and Obesity” will be a useful resource for clinicians and basic science researchers, such as biochemists, toxicologists, immunologists, nutritionists, adult and pediatric endocrinologists, cardiologists, as well as students who are thought in this field.
出版日期Book 20171st edition
关键词Adipose Tissue Hypoxia; Lipolysis; Lipotoxicity; Obesity; Protein Kinases
版次1
doihttps://doi.org/10.1007/978-3-319-48382-5
isbn_softcover978-3-319-83928-8
isbn_ebook978-3-319-48382-5Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerl
The information of publication is updating

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发表于 2025-3-21 23:14:09 | 显示全部楼层
Circadian Rhythms in Diet-Induced Obesity,trient-sensing neurons involved in the regulation of energy and glucose homeostasis. The timing of three-meal schedules indicates close association with the plasma levels of insulin and preceding food availability. Desynchronization between the central and peripheral clocks by altered timing of food
Delectable
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钱财
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Obesity, Persistent Organic Pollutants and Related Health Problems,ncapacitate detoxification of POPs. Obesity could be deemed as a multidimensional condition, as various factors interact to lead to an obesogenic result. Therefore, concomitant disorders may occur, from mild to lethal, and get intensified due to POPs exposure. POPs exact function mechanisms remain r
Morphine
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Asseverate
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Culpable
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枕垫
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What Is Lipotoxicity?, long-chain non-esterified fatty acids and their products such as ceramides and diacylglycerols (DAGs). Thus, excess DAG, ceramide and saturated fatty acids in obesity can induce chronic inflammation and have harmful effect on multiple organs and systems. In this context, chronic adipose tissue infl
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The Pathogenesis of Obesity-Associated Adipose Tissue Inflammation,through hyperactivation of c-Jun N-terminal Kinase (JNK)-Activator Protein 1 (AP1) and inhibitor of nuclear factor kappa-B kinase beta (IKKbeta)-nuclear factor kappa B (NF-kappaB) pathways play an important role, and may also affect vascular endothelial function by modulating vascular nitric oxide a
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