Titlebook: Obesity; Pathogenesis, Diagno Paolo Sbraccia,Nicholas Finer Reference work 2019 Springer Nature Switzerland AG 2019 Neuroendocrinology of e

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Obesity and NAFLD,ects normal physiology and metabolism, causing multiple associated diseases such as cardiovascular disease, diabetes, nonalcoholic fatty liver disease (NAFLD), and several types of cancer, among others. The main contributors for NAFLD development and progression include high concentrations of diet a

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Obesity and Type 2 Diabetes,e important concept of long-term reversibility of type 2 diabetes is discussed along with the beta-cell dedifferentiation, which explains the insulin secretory defect..Obesity brings about distinct pathophysiological changes as a consequence of individuals’ pattern of food intake and levels of activ

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Obesity, Hypertension, and Dyslipidemia,asma triglycerides, reducing high-density lipoprotein cholesterol, and inducing a shift to a proatherogenic composition (small, dense) of low-density lipoproteins. Obesity activates the sympathetic nervous system, increases sodium and water reabsorption, and increases the production of angiotensin I

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Obesity and Obstructive Sleep Apnea Syndrome,7% of all adults, but the risk rises with increasing body weight and it is very common in people with obesity. OSA causes symptoms of daytime sleepiness which can be disabling for patients, and this is currently the main criterion used to determine if treatment should be offered. Although obesity it

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Obesity in Children and Adolescents,olescents is highly prevalent with the rates of severe obesity, in particular, increasing considerably. Biological contributors, principally in the form of genetic risk which is susceptible to expression in the context of an obesogenic environment, account for the primary etiology of childhood obesi

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