Titlebook: Nutritional Treatment of Chronic Renal Failure; Sergio Giovannetti Book 1989 Kluwer Academic Publishers 1989 assessment.blood.diagnosis.di

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A Historical Review of Low-Protein Diets,k diet and bed rest, both of which are noxious. The former is noxious due to the high protein (PR), sodium (Na), and inorganic phosphorus (Pi) content, and the latter is noxious due to its catabolic action. Traces of this “therapeutic” philosophy were still present in the countryside in the first de

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Chronic Renal Failure: Definition, Causes, and Assessment, however, having a completely different meaning (see Chapter 5). Tubular dysfunction, or abnormalities in the composition of the glomerular filtrate, are not regarded as manifestations of .. Thus the acronym CRF indicates a chronic reduction of glomerular filtration rate (GFR) below the normal range

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Causes of Progression of Renal Disease,on to end-stage disease occurs frequently, even if the initiating event or condition is resolved or eradicated. The immunologic mechanisms and the mediators of kidney tissue damage that they induce may be responsible for initiating most renal diseases, but certain clinical and experimental observati

lipids

The Uremic Syndrome: Definition, Diagnosis, Causes of Signs and Symptoms, Complications,its duration, the symptoms of the uremic syndrome may differ greatly in nature and severity, iatrogenic factors (see Chapter 15) and therapy (see Chapters 14 and 15) may largely influence the clinical aspect of uremic patients when compared to untreated ones.

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Abnormalities of Lipid Metabolism,g subsequent dialysis and transplantation. These disturbances have been associated not only with the development of premature cardiovascular disease [1], but also, more recently, with the development of glomerulosclerosis and progression of renal disease [2].

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Abnormalities of Carbohydrate Metabolism,sponse to oral or intravenous glucose, whereas the occurrence of fasting hyperglycemia is definitely less common. Spontaneous hypoglycemia is also known to occur in patients with terminal renal failure, whether diabetic or not [2, 3]. The impaired glucose tolerance of uremia rarely poses therapeutic

Coronation

Sodium, Potassium, and Hydrogen Ions in Chronic Renal Failure,s, which appears to be geared to preserving fluid and electrolyte homeostasis. The adaptive functional changes in surviving nephrons show predictable patterns, regardless of the underlying renal disease. Exceptions to these patterns may occur when any component of the system responsible for the adap

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