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Titlebook: Nutritional Treatment of Chronic Renal Failure; Sergio Giovannetti Book 1989 Kluwer Academic Publishers 1989 assessment.blood.diagnosis.di

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发表于 2025-3-21 16:19:16 | 显示全部楼层 |阅读模式
书目名称Nutritional Treatment of Chronic Renal Failure
编辑Sergio Giovannetti
视频video
丛书名称Topics in Renal Medicine
图书封面Titlebook: Nutritional Treatment of Chronic Renal Failure;  Sergio Giovannetti Book 1989 Kluwer Academic Publishers 1989 assessment.blood.diagnosis.di
描述Enormous progress has been made in the treatment of chronic renal failure over the last decades. Until the 1950s, chronic renal failure was considered to be an inexorably lethal condition. This is no longer the case. In addition, the disease, severe uremic syndrome, is now extremely rare, if existent at all, in industrialized countries. Physicians of my generation who saw patients hospitalized with hemor­ raghes, pericarditis, severe anemia, cardiac failure, "malignant hypertension," pruritus, vomiting, generalized edema, and convulsions are particularly grate­ ful for this progress. I well remember seeing such patients hospitalized in the last days or weeks of their lives and also remember the sense of impotence I suffered for the com­ plete lack of efficient measures I had at my disposal to manage their condition. Nowadays, hemodialysis, peritoneal dialysis, and kidney transplantation allow patients with chronic renal failure to survive for very long periods of time in a satisfactory condition. Why then is there still a sense of dissatisfaction and why should we study dietary management? The drawbacks of dialysis and transplantation are the main reasons, but the certainty that di
出版日期Book 1989
关键词assessment; blood; diagnosis; dialysis; hemodialysis; hospital; hypertension; impotence; kidney; management; m
版次1
doihttps://doi.org/10.1007/978-1-4613-1583-4
isbn_softcover978-1-4612-8877-0
isbn_ebook978-1-4613-1583-4
copyrightKluwer Academic Publishers 1989
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Abnormalities of Carbohydrate Metabolism,ly characterized by the presence of tissue insensitivity to insulin. This basic cellular derangement probably causes chronic hyperinsulinism and, through the sequence hyperin-sulinemia → hyperlipidemia → accelerated atherosclerosis, bears a direct relation to the high incidence of vascular accidents in uremic patients.
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Adaptation of Remnant Nephrons,iculty, with less than 40,000 nephrons (2% of normal renal function). The challenge to survival posed by the loss of nephrons is met by a variety of adaptive mechanisms, most of which are not fully understood.
发表于 2025-3-22 07:22:23 | 显示全部楼层
Sodium, Potassium, and Hydrogen Ions in Chronic Renal Failure,tation has been specifically damaged. In this chapter we will briefly describe how the chronically failing kidney copes with the demand for sodium (Na.), potassium (K.) and hydrogen ion (H.) homeostasis.
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considered to be an inexorably lethal condition. This is no longer the case. In addition, the disease, severe uremic syndrome, is now extremely rare, if existent at all, in industrialized countries. Physicians of my generation who saw patients hospitalized with hemor­ raghes, pericarditis, severe an
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Indications and Contraindications for Nutritional Therapy in Chronic Renal Failure,rations that follow the ablation of kidney tissue. Hyperperfusion and hyperfiltration, which may lead to glomerular sclerosis (see Chapter 4) and even focal glomerulosclerosis, were found in 4 out of 24 patients nephrectomized for benign renal disease [3].
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