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Titlebook: Nutrient-Induced Responses in Eukaryotic Cells; Joris G. Winderickx,Peter M. Taylor Book 20041st edition Springer-Verlag Berlin Heidelberg

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发表于 2025-3-21 18:02:59 | 显示全部楼层 |阅读模式
书目名称Nutrient-Induced Responses in Eukaryotic Cells
编辑Joris G. Winderickx,Peter M. Taylor
视频video
概述Up-to-date status of nutrient-induced responses, presented by experts in the field
丛书名称Topics in Current Genetics
图书封面Titlebook: Nutrient-Induced Responses in Eukaryotic Cells;  Joris G. Winderickx,Peter M. Taylor Book 20041st edition Springer-Verlag Berlin Heidelberg
描述.Cells of all living organisms have the ability to respond to altered nutritional conditions. They have developed mechanisms to sense nutrient availability and to produce appropriate responses, which involve changes in gene expression and the production or degradation of certain enzymes and other proteins. In recent years, the understanding of nutrient-induced signal transduction has greatly advanced and the emerging picture is that nutrient signalling mechanisms evolved early in evolution. This book provides a detailed presentation and comparison of the key nutritional regulatory mechanisms in lower as well as higher eukaryotes, written by recognised experts in this expanding field..
出版日期Book 20041st edition
关键词Hormones; Nutrients; Signal transduction; Yeasts; amino acid; enzyme; enzymes; evolution; Expression; gene; ge
版次1
doihttps://doi.org/10.1007/b88442
isbn_softcover978-3-662-30822-6
isbn_ebook978-3-540-39898-1Series ISSN 1610-2096 Series E-ISSN 1610-6970
issn_series 1610-2096
copyrightSpringer-Verlag Berlin Heidelberg 2004
The information of publication is updating

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发表于 2025-3-21 20:31:46 | 显示全部楼层
1610-2096 ovides a detailed presentation and comparison of the key nutritional regulatory mechanisms in lower as well as higher eukaryotes, written by recognised experts in this expanding field..978-3-662-30822-6978-3-540-39898-1Series ISSN 1610-2096 Series E-ISSN 1610-6970
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3 Antagonists of the TOR pathway in animal cells, of the GAP activity of Tsc2 that functions between Tsc1/Tsc2 and TOR. These exciting developments demonstrate that . is a powerful system to decipher the architecture of the TOR signaling network at molecular, cellular and organismal levels.
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7 Regulation of the yeast general amino acid control pathway in response to nutrient stress,-directed regulation of transcription. Recent studies suggest that Gcn4p can also interface with other stress response pathways, allowing the cell to direct the timing and content of stress gene expression in response to multiple stress inputs.
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8 Tor-signaling and Tor-interacting proteins in yeast, characterization of interacting partners of Tor1p and Tor2p. These studies have converged in unanticipated ways to yield new insights into how these kinases may function both to receive as well as transmit nutritional information in yeast.
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9 Integrated regulation of the nitrogen-carbon interface,bon backbone of glutamate when the tricarboxylic acid cycle is inoperative. . Torl/2, a master cellular regulator, reported to be the critical link connecting environmental nutritional conditions with the transcriptional responses they trigger.
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