Titlebook: Non-Neuronal Mechanisms of Brain Damage and Repair After Stroke; Jun Chen,John H. Zhang,Xiaoming Hu Book 2016 Springer International Publi

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intuition

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https://doi.org/10.1007/978-3-319-32337-4blood brain barrier; brain repair; neuroinflammation; therapies; white matter

疯狂

Structural Alterations to the Endothelial Tight Junction Complex During Strokenctions (TJs) that link the cerebral endothelial cells are critical for BBB function and they undergo marked modifications after stroke. Those changes contribute to increased BBB permeability, brain edema, and leukocyte infiltration, all hallmarks of stroke. This review describes the types of TJ mod

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Role of Pericytes in Neurovascular Unit and Stroke that a successful neuroprotection is not feasible without microvascular protection. Recent studies on pericytes, long-neglected cells of the NVU have provided insight into the regulation of microcirculatory flow, blood–brain barrier (BBB) maintenance and angiogenesis. Ischemia induces persistent pe

虚构的东西

Glial Support of Blood–Brain Barrier Integrity: Molecular Targets for Novel Therapeutic Strategies istics (i.e., tight junction protein complexes, influx and efflux transporters) that control permeation of circulating solutes including drugs. In order to form this “barrier,” brain microvascular endothelial cells require support of adjacent astrocytes and microglia. This intricate relationship also

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雪崩

Angiogenesis: A Realistic Therapy for Ischemic Strokeo various organs and tissue. Angiogenesis is strictly regulated by many key anti- or pro-angiogenic factors, and these molecules have been well-documented to play an important role in the development of angiogenesis in response to various pathological conditions. In human diseases angiogenesis has a