Titlebook: Nitrates Updated; Current Use in Angin Udho Thadani (Professor of Medicine, Vice Chief Ca Book 1997 Kluwer Academic Publishers 1997 angina

VOK

Mechanisms of Action of Nitrates,myocardial infarction, and congestive heart failure. Organic nitrate esters have a direct relaxant effect on vascular smooth muscles, and the dilation of coronary vessels improves oxygen supply to the myocardium. The dilation of peripheral veins, and in higher doses peripheral arteries, reduces prel

explicit

Mechanisms of Nitrate Tolerance, with stable angina pectoris and congestive heart failure. Classical hypotheses of nitrate tolerance suggest the phenomenon to result from vascular depletion of critical sulfhydryl groups, which are necessary to bring about vasorelaxation from nitrates. While this mechanism of nitrate tolerance prob

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Clinical Pharmacokinetics of Nitrates,lasma concentrations is very difficult. Its pharmacokinetics are unusual, with a very rapid disappearance from plasma, and large intraindividual and interindividual variations. After oral administration, there seems to be a very extensive first-pass hepatic extraction and the plasma concentrations a

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Nitrates for Unstable Angina, with or without additional thrombus formation, is invariably responsible for a prolonged episode of angina at rest, increasing frequency of angina at rest, or with minimal exertion of less than 4 weeks in duration and early postinfarction angina. Plaque progression, rather than plaque fissuring, is

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残暴

The Organic Nitrates: Practical Problems In Their Use,nduce dizziness or presyncope. Cerebral vasodilatation produces headaches. Nitrate tolerance is probably multifactorial, and results in attenuation or loss of nitrate action. Tolerance is well documented in angina pectoris and congestive heart failure. The only proven strategy to prevent tolerance i