Titlebook: New Aspects in the Treatment of Failing Heart; Hisakazu Yasuda (Emeritus Professor),Hideaki Kawag Conference proceedings 1992 Springer-Ver

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Christophe Leclercq,Jean-Marie Moalic,Daniéle Charlemagne,Pascale Mansier,Claude Sainte-Beuve,Franco

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Hideaki Kawaguchi,Mikako Shoki,Hitoshi Sano,Toshiyuki Kudo,Hirofumi Sawa,Hiroshi Okamoto,Naoki Mochi

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Postischemic Myocardial Dysfunction (Myocardial “Stunning”): Role of Oxidative StressIn addition, coronary reperfusion by means of thrombolytic therapy, percutaneous transluminal angioplasty or bypass surgery is rapidly emerging as the fundamental strategy in the management of acute ischemic syndromes. The recent appreciation of the role of reperfusion in the natural history of coro

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Degradation of Membrane Phospholipids in the Ischemic and Reperfused Heartlactate to fulfil their energy requirements. Contraction and relaxation of the myofibrils consume over 70 of ATP produced by mitochondrial activity, the remaining part is used for transport of ions across membranes, and maintenance of cellular integrity. The plasmalemma, the membrane enclosing the c

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Role of Protein Kinase System in the Signal Transduction of Stretch-mediated Myocyte Growthshes and imposed an in vitro mechanical load by stretching the adherent cells. Myocyte stretching increased total cell RNA content and mRNA levels of c-fos and skeletal α-actin followed by activation of protein synthesis. CAT assay indicated that sequences containing a serum response element were re

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PI Response and Calcium Overload in Cardiomyopathic Hamster Heart Cellosphatidylinositol (PI) turnover pathway generates two second messengers, inositol-(l,4,5)-trisphosphate (IP.) and sn-1,2-diacylglycerol (DAG) (5,6). DAG stimulates membrane-bound phospholipid-dependent, Ca.- dependent protein kinase C (7), while IP. releases Ca. from stores in the sarcoplasmic reti