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Titlebook: Neuroprotection and Regeneration of the Spinal Cord; Kenzo Uchida,Masaya Nakamura,Yoshiaki Toyama Book 2014 Springer Japan 2014 OPLL.biome

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楼主: 迅速
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Spinal Kyphosis Causes Demyelination and Neuronal Loss in the Spinal Cord kyphosis. In the past, no animal model to clarify the mechanism of spinal cord damage due to spinal kyphotic deformity has been available. We developed a new experimental model of chronic spinal cord compression using a bird, to evaluate the effects of chronic compression associated with kyphotic d
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Granulocyte Colony-Stimulating Factor-Mediated Neuroprotective Therapy for Spinal Cord Injury SCI models. Through those experiments, we showed G-CSF’s mechanisms of action for SCI..G-CSF showed efficacy for SCI through mobilization of bone marrow-derived cells. G-CSF attenuated neuronal cell death in vitro and in vivo, resulting in promotion of functional recovery after SCI. Expression of I
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The Proteoglycan-Degrading Enzymes Promote Functional Recovery After Spinal Cord Injury: Keratan Sulhibitory cues. Chondroitin sulfate (CS) proteoglycans are strong inhibitors of neuronal injuries of the adult CNS. Besides CS chains, keratan sulfate (KS) chains are also covalently attached to some proteoglycans. However, the biological significance of KS remains to be not well understood. Here we
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Targeted Retrograde Gene Delivery into the Injured Spinal Cord Using Recombinant Adenovirus Vector Curotrophic factor (BDNF) gene for the rescue of injured rat spinal cord. AdV-BDNF gene or AdV-β-galactosidase (AdV-LacZ) gene was injected into the sternomastoid muscle immediately after traumatic C4 spinal cord injury. Localization of β-galactosidase expression produced by LacZ gene or AdV-BDNF gen
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nagement. A number of recent events underscore this development. In February 2000 the Indian Institute of Management organized a “Corporate Reputation for Competitive Advantage” workshop in Calcutta, which focused on spirituality, ethics and leadership. The conference “Business, Religion and Spiritu
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