Titlebook: Neurochemical Mechanisms in Disease; John P. Blass Book 2011 Springer Science+Business Media, LLC 2011

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Monoamine Transporter Pathologies,responsible for the termination of synaptic signaling by removing neurotransmitters from the synaptic cleft. Altered monoaminergic transporter function has been implicated in the pathology of disease states such as depression , anxiety , addiction, autism, Parkinson’s disease , and attention deficit

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Glutamate and Glutamine in Brain Disorders,diseases and improved pharmacotherapy is of significant value. In this respect, knowledge on malfunctions of metabolic homeostasis related to the neurotransmission process is still limited. As evident from this chapter, failure of the metabolic homeostasis of the two amino acids of major importance,

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Rho-Linked Mental Retardation Genes,extremely heterogeneous, including environmental factors as well as genetic changes, such as chromosomal abnormalities and single-gene mutations. Great progress has been made in recent years towards the identification of MR genes, particularly X-linked MR genes. A largely remaining challenge, howeve

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,NF-κB in Brain Diseases,. NF-κB transcription factors are ubiquitously expressed, including neurons and glial cells of the central nervous system (CNS). The roles of NF-κB family in the CNS, both as mediators of transcriptional response to synaptic activity and in behavioral paradigms of learning and memory, are the focus

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Trinucleotide-Expansion Diseases, genome. In many cases, the longer the repeat the earlier the onset, and the more rapid and severe is the disease progression. Almost all of these diseases may be divided into three groups. In the first group, the expansion is either in an untranslated region/intron of the gene (e.g., fragile X synd

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CNS Cytokines, vascular permeability is increased and leukocyte extravasation occurs. It is now realized that the central nervous system (CNS) is not as devoid of immune cell entrance as once believed and that neuroinflammation can occur. Even in the CNS absence of peripheral immune cells, cytokines from the peri

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RNA Pathologies in Neurological Disorders,nctional roles and regulations of noncoding RNAs including antisense RNAs and small RNAs are in progress. In addition, the human genome project revealed that we humans carry as few as ∼22,000 genes. Humans exploit tissue-specific and developmental stage-specific alternative splicing to generate a la