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Titlebook: Nephrotoxic Mechanisms of Drugs and Environmental Toxins; George A. Porter Book 1982 Springer Science+Business Media New York 1982 Ketone.

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grates biology, chemistry, and physics for a multidisciplina.The volumes in this authoritative series present a multidisciplinary approach to modeling and simulation of flows in the cardiovascular and ventilatory systems, especially multiscale modeling and coupled simulations. The cardiovascular and
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Pathology of Acute Renal Failurer we will describe and discuss, with an emphasis on structure-function correlations, a major subset of the clinical syndrome of ARF, that is, acute tubular necrosis (ATN). By definition, the term ATN includes all forms of acute urinary suppression in which tubular injury of acute onset is the primar
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Heavy Metal Models of Experimental Acute Renal Failureof experimental ARF is a reflection both of the search for the “perfect” model as well as the heterogeneity of clinical ARF. In general, the experimental models of ARF may be subdivided into those induced with a nephrotoxin and those following a period of renal ischemia (Flamenbaum, 1973). A similar
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Studies on Segmental Transport in Models of Acute Renal Failure this syndrome is based on animal models using a host of modalities to precipitate the renal failure and an equally impressive list of maneuvers to ameliorate or protect agar (***) ARF. Renal artery clamping for 60 min is a model we have (***) to examine the pathophysiology of this syndrome at the l
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Antibiotic Nephrotoxicitynephrotoxin-induced acute renal failure was 9% (Levinsky and Alexander, 1976), which in the last decade has risen to over 15% in series reporting consecutively occurring cases (McMurray ., 1978; Anderson ., 1977; Galpin ., 1978). Antibiotics continue as the most frequently cited etiology of nephroto
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