Titlebook: NADPH Oxidases; Methods and Protocol Ulla G. Knaus,Thomas L. Leto Book 2019 Springer Science+Business Media, LLC, part of Springer Nature 2

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Fiona Augsburger,Aleksandra Filippova,Vincent Jaquethat can be accomplished within a given energy budget will eventually require increasing the degree to which our computing technologies avoid information loss, ., are logically reversible. But the traditional definition of logical reversibility is actually more restrictive than is necessary to avoid

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Balázs Enyedi,Miklós Geiszthat can be accomplished within a given energy budget will eventually require increasing the degree to which our computing technologies avoid information loss, ., are logically reversible. But the traditional definition of logical reversibility is actually more restrictive than is necessary to avoid

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Intersecting Stories of the Phagocyte NADPH Oxidase and Chronic Granulomatous Diseaseettings. Optimal oxidant-dependent antimicrobial activity by neutrophils relies on the ability of stimulated phagocytes to utilize a multicomponent NADPH oxidase to generate oxidants. The frequent, severe, and often fatal infections experienced by individuals with chronic granulomatous disease (CGD)

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Mammalian NADPH Oxidasesticular), which consistently lead to aging and disease. Over the last decades, however, it became increasingly apparent that virtually all eukaryotic cells possess specifically ROS-producing enzymes, namely, NOX NADPH oxidases. In most mammals, there are seven NOX isoforms: three closely related iso

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NOX5 Cell-Free Assay for the High-Throughput Screening of Small Moleculesenriched in the phagocyte NOX isoform (NOX2). This feat has allowed the development of a complex NOX2 cell-free assay, which has been a key tool for the understanding of the mode of action of NOX2, its biochemistry, pharmacology, and identification of NOX2-specific inhibitors. In addition to NOX2, t