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Titlebook: Metabolism and Artificial Nutrition in the Critically Ill; G. Guarnieri,F. Iscra Book 1999 Springer-Verlag Italia 1999 Enteral nutrition.I

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发表于 2025-3-21 16:40:44 | 显示全部楼层 |阅读模式
书目名称Metabolism and Artificial Nutrition in the Critically Ill
编辑G. Guarnieri,F. Iscra
视频video
丛书名称Topics in Anaesthesia and Critical Care
图书封面Titlebook: Metabolism and Artificial Nutrition in the Critically Ill;  G. Guarnieri,F. Iscra Book 1999 Springer-Verlag Italia 1999 Enteral nutrition.I
描述The critically ill patient in intensive care may present with serious metabolic alterations caused directly by the illness or secondarily by complications (e.g. infections, organ failure or sepsis) developing within a few hours of hospitaliza­ tion or in the following days. Among the situations which maintain and further trigger rapidly evolving altered metabolism are complex hormonal reactions, particularly those of the hypothalamus-hypophysis-adrenal axis, and abnormal stimulation of the autonomic nervous system. In fact, the sympathetic nervous system is known to cause significant metabolic alterations. For example, a surgery patient afflicted by septic complications may become hypercatabolic and experience significant nitrogen loss; the altered protein metabolism may in turn heavily influence carbohydrate and lipid metabolism as well. Thus, it is apparent that for optimal care of patients with altered metabolic functions, further knowledge is necessary regarding the physiopathology of metabolism and the physiopathological mechanisms, which alter the consump­ tion of principal energy substrates. Many experimental and clinical studies have investigated the metabolic aspects of in
出版日期Book 1999
关键词Enteral nutrition; Insulin; Parenteral nutrition; Sepsis; Trauma; artificial nutrition; body composition; c
版次1
doihttps://doi.org/10.1007/978-88-470-2901-9
isbn_softcover978-88-470-0042-1
isbn_ebook978-88-470-2901-9
copyrightSpringer-Verlag Italia 1999
The information of publication is updating

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Topics in Anaesthesia and Critical Carehttp://image.papertrans.cn/m/image/631271.jpg
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Which metabolic strategies in the early phase of injury?The host’s response to tissue injury, commonly called trauma reaction, appears to bea spectrum within which one can identify several patterns. With a single uncomplicated injury, the response peaks between days 3 and 5 post-injury and decreases within between 7 and 10 days.
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Muscle catabolism, amino acid flux and protein turnover in injury of acute protein catabolism include impaired immune response to infections, decreased coagulation capacity, impaired wound healing, reduced gut function and muscle wasting. In some patients weakness of respiratory muscles may be so great as to impair pulmunary ventilation and contribute to the respiratory insufficiency associated with trauma.
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Effects of artificial nutrition on the immune system cellsgies (burns, major surgery, trauma) (Table 1). Moreover, other conditions in injured patients may further worsen immunosuppression, such as bleeding, transfusions, sepsis, protein-calorie malnutrition, side effects of anesthesia and drugs.
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Metabolism and Artificial Nutrition in the Critically Ill
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Metabolism and Artificial Nutrition in the Critically Ill978-88-470-2901-9
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