Titlebook: Medullary Thyroid Carcinoma; Biology – Management Friedhelm Raue Book 2015 Springer International Publishing Switzerland 2015 Calcitonin.Me

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Thyroid C-Cell Biology and Oncogenic Transformation, receptor is targeted primarily to the C-cell, providing one possible cause of tissue-specific oncogenesis. The role of RET signaling in normal C-cell function is unknown though calcitonin gene transcription appears to be sensitive to RET activation. Beyond RET, the modeling of oncogenesis in animal

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Pheochromocytomas in Multiple Endocrine Neoplasia Type 2,onal techniques of . I-metaiodobenzylguanindine (MIBG) scintigraphy, computer tomography and magnetic resonance imaging. Treatment of PC is surgical and requires peri-operative alpha and, frequently, beta blockade. Novel surgical techniques, such as adrenal sparing surgery and a laparoscopic approac

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Long-Term Follow-up in Medullary Thyroid Carcinoma, When the postoperative serum CTN level exceeds 150 pg/ml, patients should be evaluated with imaging procedures, including computed tomography (CT) of the neck and chest, contrast-enhanced magnetic resonance imaging (MRI) and ultrasound (US) of the liver, bone scintigraphy, MRI of the bone, and posi

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