Titlebook: Mechanosensitivity of the Heart; Andre Kamkin,Irina Kiseleva Book 2010 Springer Science+Business Media B.V. 2010 Activation.Angiotensin II

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Mechanical Stretch-Induced Reorganization of the Cytoskeleton and the Small GTPase Rac-1 in Cardiac seen during neonatal development or in the hypertensive adult promote a fibrotic response in the heart. In vitro studies have established that mechanical stretch of isolated cardiac fibroblasts directly stimulates expression of extracellular matrix components and proliferation, both hallmarks of fib

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Molecular Signaling Mechanisms of Myocardial Stretch: Implications for Heart Diseaseation, remodeling and gene expression. Mechanical load is a major cause of cardiac hypertrophy. Since the initial observation of stretch-induced growth, our understanding of this complex field has been steadily growing, but remains incomplete. The mechanisms by which myocardial cells convert mechani

apiary

Mechanical Stress Induces Cardiomyocyte Hypertrophy Through Agonist-Independent Activation of Angiotoad-induced cardiac hypertrophy. Besides systemically and locally generated AngII, mechanical stress can activate the AT. receptor, and induce cardiac hypertrophy in vivo. In response to stretch stimulation, the AT. receptor undergoes a specific switch in the receptor conformation without the involv

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Mechanostransduction in Cardiac and Stem-Cell Derived Cardiac Cellsinclude: methods to apply mechanical stimuli to isolated cells and tissues; methods for patterned growth of cells; effects of stretch and shear stress on cellular function and tissue electrophysiology; regulation of structural and junctional proteins by stretch; the role of the cytoskeleton in mecha

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Effects of Applied Stretch on Native and Recombinant Cardiac Na+ Currentsical conditions the mechanical activity of the heart may change tissue excitability, the action potential waveform and/or the pattern of conduction. In some cases, this mechanoelectrical feedback can alter the myocardium such that extrasystoles or rhythm disturbances are observed. It is thought that

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Mechanosensitive Alterations of Action Potentials and Membrane Currents in Healthy and Diseased Cardchanges in mechano-gated channels, mechanosensitive whole-cell currents which lead to membrane depolarization which is equivalent to a decrease in the resting membrane potential and elicited stretch-induced depolarizations, that appear during repolarization phase of cardiomyocyte action potential. S

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The Role of Mechanosensitive Fibroblasts in the Heart: Evidence from Acutely Isolated Single Cells, tential. These changes of fibroblasts membrane potential are determined by operation of mechano-gated channel (MGCs). Two types of MGCs with conductances of 43 pS and 87 pS were observed during direct deformation of the fresh isolated cells. Cell compression augment the whole-cell MG currents and in

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