Titlebook: Mechanisms of Secondary Brain Damage from Trauma and Ischemia; Recent Advances of o A. Baethmann,J. Eriskat,N. Plesnila Conference proceedi

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书目名称Mechanisms of Secondary Brain Damage from Trauma and Ischemia影响因子(影响力)




书目名称Mechanisms of Secondary Brain Damage from Trauma and Ischemia影响因子(影响力)学科排名




书目名称Mechanisms of Secondary Brain Damage from Trauma and Ischemia网络公开度




书目名称Mechanisms of Secondary Brain Damage from Trauma and Ischemia网络公开度学科排名




书目名称Mechanisms of Secondary Brain Damage from Trauma and Ischemia被引频次




书目名称Mechanisms of Secondary Brain Damage from Trauma and Ischemia被引频次学科排名




书目名称Mechanisms of Secondary Brain Damage from Trauma and Ischemia年度引用




书目名称Mechanisms of Secondary Brain Damage from Trauma and Ischemia年度引用学科排名




书目名称Mechanisms of Secondary Brain Damage from Trauma and Ischemia读者反馈




书目名称Mechanisms of Secondary Brain Damage from Trauma and Ischemia读者反馈学科排名

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Lessons from epidemiologic studies in clinical trials of traumatic brain injuryTirilizad Trials which enrolled over 1,100 patients. A major observation from that trial was that pretreatment hypotension or hypoxia could be unbalanced even in a large two arm blinded study. Another issue of the tirilazad trial was the influence of gender affecting not only outcome but also drug metabolism.

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Pharmacological preconditioning in global cerebral ischemia function. Bcl-2-protein is expressed in neurons that survive cerebral ischemia and may parallel the time course of tolerance after ischemic preconditioning. With this study we examined whether differences in bcl-2-protein expression compared to baseline may be involved in the induction of ischemic tolerance using 3-NPA.

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