Titlebook: Mechanisms of Physical and Emotional Stress; George P. Chrousos,D. Lynn Loriaux,Philip W. Gold Book 1988 The Editor(s) (if applicable) and

容易生皱纹

Studies on the Secretion of Corticotropin-Releasing Factor into Hypophysial Portal Bloodhe ACTH-regulatory factors include corticotropin-releasing factor (CRF), arginine-vasopressin (AVP), oxytocin, epinephrine and angiotensin II.. There is now evidence that CRF is the major physiologic regulator of ACTH secretion.. Therefore, the measurement of CRF release into hypophysial portal bloo

有特色

Is There a “Final Common Pathway” in the Regulation of ACTH Release?leasing factor (CRF). and vasopressin (AVP).. The two substances have at least additive, if not synergistic effects in stimulating ACTH release from the anterior pituitary .. and ... Stimulated ACTH release . can be inhibited by the administration of antisera or antagonists to either CRF. or AVP.. D

舔食

Regulation of Vasopressin and Oxytocin Release in vitroOT release potentiate the effect of corticotropin releasing factor (CRF) on ACTH release (1), and they are present in portal plasma at the concentration required for this action (2). VP and CRF are co-localized in parvicellular neurons in the paraventricular nucleus which project to the median emine

忘川河

Regulation of Rat Hypothalamic Corticotropin-Releasing Hormone Secretion ,: Potential Clinical Implist that CRH not only stimulates pituitary adrenocorticotropin (ACTH) secretion (3), but also activates the sympathetic nervous system (4) and causes behavioral changes (5–10). Behavioral studies conducted primarily in rats have indicated that CRH administered intracerebroventricularly (icv) causes a

ensemble

Is the Benzodiazepine/GABA Receptor Chloride Ionophore Complex Involved in Physical and Emotional Stsal relationships between specific receptor/effector systems in the central nervous system (CNS) and stress-induced somatic, endocrine and behavioral responses have not been firmly established, both physical and emotional stressors have been shown to affect a variety of neurotransmitters, neuromodul

惊呼

Corticosteroids and the Immune Responseciple, one could develop the theme; stress → steroid production → immune modulation. In practice, however, one should recognize that most of our knowledge of the effects of glucocorticosteroids on the immune response comes from pharmacological studies where exogenous steroids are given and where the

FEIGN

Lipocortinsienes and PAF-acether) always involve a membrane enzyme activation namely phospholipase A.. This activation leads to the cleavage of the phospholipids present in the membrane and permits the release of an unsaturated fatty acid from the 2 position - usually, arachidonic acid - and to the simultaneou

chassis

鸣叫

Is the Benzodiazepine/GABA Receptor Chloride Ionophore Complex Involved in Physical and Emotional Stators, second messenger systems, and their associated recognition sites in the CNS (this volume and [1] for overviews). Thus, it is not surprising that most schemata describing stress-induced alterations in the CNS focus on the hypothalamus, although both the control and generation of stress must be initiated in higher centers.

哑巴

Book 1988 the sympathetic system was associated with Walter Cannon‘s "fight or flight" response. The intervening years have witnessed a number of dis­ coveries that have furthered our understanding of the mechanisms of the stress response. The isolation, identification and manufacture of gluco­ corticoids, t