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Titlebook: Mechanisms in B-Cell Neoplasia 1990; Workshop 1990 at the Michael Potter,Fritz Melchers Book 1990 The Editor(s) (if applicable) and The Aut

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书目名称Mechanisms in B-Cell Neoplasia 1990
副标题Workshop 1990 at the
编辑Michael Potter,Fritz Melchers
视频video
丛书名称Current Topics in Microbiology and Immunology
图书封面Titlebook: Mechanisms in B-Cell Neoplasia 1990; Workshop 1990 at the Michael Potter,Fritz Melchers Book 1990 The Editor(s) (if applicable) and The Aut
描述The eighth workshop in this series on Mechanisms in B-Cell Neoplasia 1990 was held in Wilson Hall at the National Institutes of Health, Bethesda, Maryland on March 28-30. Five major topics formed the basis for the discussions: 1) progress in experimental models of B-cell tumorigenesis, 2) the role of IL-6 in plasma cell tumor formation with particular emphasis on human myeloma, 3) immortaliza­ tion and regulation of mitosis in B-cells, 4) the mYQ gene in B-cell neoplasia, and 5) the role of EBV and other oncogenes in transforma­ tion of human B-Iymphocytes. A meeting on the Epidemiology of Myeloma was held at the N. I. H. on the preceding day, and many of those interested in the clinical aspects of myeloma were also participants at the workshop. Experimental Models of B-Cell Tumor Development We have seen in the last eight years the steady growth of model experimental systems, many of which have been designed to be counter­ parts of the major forms of human B cell tumors, e. g. , follicular lymphomas, Burkitt‘s lymphomas, acute B-cell leukemia and multiple myeloma. A variety of novel ways of inducing these tumors has been described. Advantage has been taken of the "experiments in n
出版日期Book 1990
关键词Burkitt‘s Lymphoma; Lymphome; Myelome; Neoplasie; Onkologie; Tumoren; oncology; plasma cells; tumorigenesis;
版次1
doihttps://doi.org/10.1007/978-3-642-75889-8
isbn_softcover978-3-642-75891-1
isbn_ebook978-3-642-75889-8Series ISSN 0070-217X Series E-ISSN 2196-9965
issn_series 0070-217X
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE
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