Titlebook: Mammary Tumorigenesis and Malignant Progression; Advances in Cellular Robert B. Dickson,Marc E. Lippman Book 1994 Springer Science+Business

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sinoatrial-node

Modulations of the epithelial phenotype during embryogenesis and cancer progressionells must modify their adhesive status and change their cytoskeletal organization. Interestingly, such modifications of cell adhesion and communication systems have been shown to occur during embryogenesis and particulary during migratory process of epithelial-mesenchymal transition (EMT). These emb

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Cell-cell adhesion in invasion and metastasis of carcinomasnd cell-to-substrate interactions. The process is initiated by disaggregation of invasive cells from the primary carcinoma — a step that requires a breakdown of intercellular adhesion (see below for a detailed discussion). Invasion then depends on novel adhesive interactions with extracellular matri

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Fetal-like fibroblasts: Their production of migration-stimulating factor and role in tumor progressierred to as . [.,.]. In spite of the general acceptance of this model, little information is currently available regarding either the frequency at which initiating genetic lesions occur or the proportion of initiated cells that proceed on to develop into a clinically recognizable malignancy. Our ass

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form of it, maghemite (γ- Fe.O.), and impurity-substituted magnetite (titanomagnetite) have on occasion been identified in biomagnetic systems, magnetite continues to be the primary magnetic source in biology. It is of interest, therefore, to inquire into the origin of its magnetism, or more properl

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Thomas J. Anderson,William R. Millerpredates both the discovery of biogenic magnetite in many species and the generation of convincing behavioral evidence for the existence of a magnetic sense. In 1945, Ising unsuccessfully searched for remanence in the head of a swallow, and Griffin (1944) noted the absence of any known magnetic mate

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