Titlebook: Male Sterility and Motility Disorders; Etiological Factors Samir Hamamah,François Olivennes,René Frydman Book 1999 Springer-Verlag New Yor

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nest investiga­ tors in the field. Standards were immensely high throughout, and discussions were meaningful and detailed. Analyses on disorders in sperm motility de­ mand a broad-based approach, involving cytologists, geneticists, andrologists, and embryologists, because the topic has many clinical

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Metabolic Strategy in Human Spermatozoa: Its Impact on Sperm Motilityproteins of the flagellum. The ability of the spermatozoon to maintain its motility pattern in vitro and in vivo, therefore, is going to depend both on the normal function of the contractile proteins of the flagellum and its ability to generate ATP but also on the availability of metabolic substrate in the female reproductive tract to produce ATP.

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Motility Updateause the main function of this cell is to deliver the male nuclear package to the ovocyte. These cases of infertility can be treated by subzonal insemination or intracytoplasmic injection (2-4), with the risk that the genetic defect will be transmitted to the offspring.

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Clinical Aspects of ICSI with Immotile Spermfrom Table 11.1, fertilization fails in half of these cycles because of a given sperm factor (i.e., no motile spermatozoon available for injection) (38% of failed cycles). On the other hand, fertilization rates may also be reduced when immotile sperm are used for ICSI (Table 11.2).

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In Vivo Therapies for Asthenozoospermiaed motility from akinethozoospermia to sliding spermatozoa and lowered sperm motility.Impaired motility because of such ultrastructural defects should be considered as congenital because they arise during the development of the sperm, essentially throughout spermiogenesis; in such cases, no in vivo treatment will offer improvement in motility.

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