Titlebook: Lymphocyte Trafficking in Health and Disease; Raffaele Badolato,Silvano Sozzani Book 2006 Birkhäuser Basel 2006 Chemokine.NK cells.T cell.

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Mystic

Lymphocyte migration to the kidneynd lead to the discovery of gene-based therapeutic options. It is likely that selective intervention of chemokines, at the appropriate phase of a particular disease, may have the therapeutic potential for site- and phase-specific intervention of lymphocyte migration and the progression of renal diseases.

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Agnosia

1422-7746 covery of chemokines and of chemokine receptors it has become evident that expression of chemokines at the site of inflammation may regulate the composition of cellular infiltrate, thereby directing the type of immune response. Recently, the molecular characterization of inherited disorders of immun

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Migration of dendritic cell subsetserable array of non-chemokine chemotactic signals and adhesion molecules in this complex process. A better understanding of the signals involved in the migration of DC subsets . constitutes a valuable basis for the development of new strategies for the control of DC migration and function under pathological conditions.

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Flinch

Controlling leukocyte trafficking in diseasetrials. The additional efforts that biologists and medicinal chemists are now placing on promiscuous small molecule antagonists should only increase the potential for market therapeutics that control human disease by controlling leukocyte trafficking.

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Migration of dendritic cell subsetszation to both peripheral non-lymphoid tissues and secondary lymphoid organs. In the absence of correct tissue localization, DC fail to promote proper immune responses. DC trafficking includes the interaction with both blood and lymphatic endothelium and the response to chemotactic signals. In the p

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