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Titlebook: Liver and Environmental Xenobiotics; S. V. S. Rana,K. Taketa Book 1997 Springer-Verlag Berlin Heidelberg 1997 Stress.drug.liver.metabolism

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Akira Ichiharaungstendenzen immun scheinen und trotz oder eben gerade wegen des verstärkten externen Drucks noch keinen Systemwechsel eingeleitet haben. Mehr als eine Dekade nach der Auflösung des sozialistischen Lagers und trotz einer latenten Wirtschaftskrise hat Kuba bisher alle Prognosen über ein unmittelbar
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Liver Injury and Serum Hyaluronan,rmful effects. In the liver, most HA is produced by Ito cells (fat-storing cells, lipocytes, stellate cells) located in the Disse’s spaces [1]. On the other hand, HA in the tissue enters the blood stream through the lymph, and is rapidly taken up via receptors into hepatic sinusoidal endothelial cells (SEC), where degradation follows [2, 3].
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Book 1997nd agricultural chemicals. Xenobiotics entering the animal will undergo one or more of the following fate: (a) elimination unchanged, (b) metabolism by enzymes, (c) spontaneous chemical transformation and (d) remain unchanged in the body. The actions of xenobiotics on the body exhibit certain specif
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dustrial and agricultural chemicals. Xenobiotics entering the animal will undergo one or more of the following fate: (a) elimination unchanged, (b) metabolism by enzymes, (c) spontaneous chemical transformation and (d) remain unchanged in the body. The actions of xenobiotics on the body exhibit cert
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Role of Nitric Oxide (NO) in the D-Galactosamine-Induced Liver Injury,o plays a role in signal transduction within the central nervous system [14]. Furthermore, NO has been shown to exert various effects on hepatocytes such as the inhibition of both protein synthesis [15, 16] and mitochondrial respiration [17, 18], and an alteration of cytochrome P-450 enzyme activity [19–21].
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Standard Liver Function Tests and Their Limitations: Selectivity and sensitivity of individual seru capacity of the liver as measured by aminopyrine clearance (aminopyrine breath test) [18, 103, 149] galactose elimination [103, 125] and lidocaine disposition [19, 118]. The third group includes tests concerned with the capacity of the liver to synthesize certain serum proteins such as albumin [154] and clotting factors.
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Oxidative Stress and Liver Injury by Environmental Xenobiotics,ly formed in all living organisms could be responsible for lipid peroxidation, leading to a sequence of chain of reactions. Therefore, oxidative stress so far remains to be a very attractive hypothesis for explaining many diseases and drug induced toxicity.
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