Titlebook: Liver Diseases in Infancy and Childhood; Samuel R. Berenberg Book 1976 Martinus Nijhoff Medical Division - The Hague 1976 childhood.fetus.

rheumatism

Bile Acid Sulfation and Cholestasis,patic parenchymal cell, the biliary ducts or both. Regardless of the etiology or the site of the lesion, cholestasis produces profound alterations in bile acid metabolism with elevated concentrations of these compounds in peripheral blood and tissues.

预兆好

玩笑

烦忧

Neonatal Susceptibility to MHV3 Infection in Mice,r example, several viral infections — e.g. herpes virus — lead to the development of severe disease contrasting with the benign form presented by normal adults. In addition, for reasons which are still unknown, newborns often developed persistent viral infections. In mice, it has been shown that age

congenial

Portal Hypertension in Early Childhood: Etiology and Diagnostic Procedures,e space limitations exclude any discussion of children with portal hypertension resulting from primary hepatic disease. It seems appropriate to limit the discussion in this manner since other sections of this book are concerned with the surgical aspects of portal hypertension and most of the surgica

可卡

随意

otic-capsule

Pathologic Liver Changes in Alpha-1-Antitrypsin Deficient Infants (Pi ZZ Phenotype),Since the first report of Freier, Sharp and Bridge in 1968, many authors have stressed the association of alpha-1-antitrypsin deficiency and liver disease in children. Aagenaes, in 1972, was the first to emphasize the frequency of prolonged neonatal cholestasis.

CHOP

Paleontology

Liver Replacement in Children,In the 10½years between the spring of 1963 and the fall of 1974, liver transplantation was attempted in 93 patients by removing the diseased native liver and replacing it with a cadaveric homograft in the natural (orthotopic) location. Fifty-six of the liver recipients were 18 years old or younger; the other 37 were adults.