Titlebook: Lipoprotein Subfractions Omega-3 Fatty Acids; H. U. Klör Conference proceedings 1989 Springer-Verlag Berlin Heidelberg 1989 atherosclerosi

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Ultracentrifugal, Chromatographic and Electrophoretic Techniques for Lipoprotein Subfractionationdescribed, and only representative applications will be cited. Also, since immunoaffinity is the topic of other presentations at this meeting, this most important methodology is omitted. Isoelectric focusing of lipoproteins, however, is described in some detail, since new results from our laboratory

Arroyo

Analysis of Lipoprotein Particles Using Immunoaffinity Chromatography and ELISA TechniquesThey are structural components for lipoprotein stability, but also co-factors for enzymes and binding ligands for cellular receptors. This has led to the use of apolipoproteins as specific markers for lipoprotein species classification. Following this concept, proposed 20 years ago by Pierre Alaupov

PAEAN

Lipoprotein Association and Function of Apolipoprotein A-IVn plasma and in interpretation of its physiologic relevance compared with that of other apolipoproteins. Although reported to occur mainly free in human plasma unassociated with lipoproteins, its amphiphilic character as an apoprotein was demonstrated very early [2, 36]. It was also shown that the g

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BAN

Lecithin: Cholesterol Acyl Transferase, Cholesterol Ester Exchange/Transfer Protein, and Lipoproteine content of CE in plasma high positively correlates with the incidence of atherosclerosis and myocardial infarction, there has been in the past and still is great interest in investigation of the enzymes involved in lipoprotein metabolism. This overview summarizes some general features, with partic

Estrogen

Role of the Liver in the Metabolism of Lipoprotein Particleslipoprotein catabolism. Most of the apolipoproteins, as well as the cholesterol and cholesterylester moieties of all circulating plasma lipoproteins, are catabolized in the liver. This makes sense because the liver is the only organ capable of degrading substantial amounts of cholesterol. The result

Herpetologist

DAFT

HDL Subclasses and Atherosclerosis[1], Gofman et al. [2], and Nikkila [3]. The second period starts with the report of Miller and Miller [4]. Since then a large amount of data have stressed the inverse relationship existing between the level of HDL and the prevalence of ischemic heart disease (IHD) [5,6].

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易发怒

Significance of the Interaction Between Lipoprotein Subfractions and Macrophages for Reverse Cholestding steroidogenesis, bile salt formation, cholesterol uptake, the binding of cholesterol ester transfer protein (CETP), and reverse cholesterol transport. The precursors of plasma HDL originate from the intestine or the liver, and upon formation of HDL. particles, tissue cholesterol is generated an