Titlebook: Lipid Metabolism in the Healthy and Disease Heart; Ger J. Vusse,Hans Stam Book 1992 Springer Science+Business Media Dordrecht 1992 ATP.Lip

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Myocardial fatty acid oxidation during ischemia and reperfusion,tially toxic intermediates such as acylCoA and acylcarnitine. After reperfusion both myocardial oxygen consumption and fatty acid oxidation may rapidly recover to preischemic levels, even when contractile function remains depressed. The mechanisms underlying the apparent dissociation between contrac

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The relative contribution of glucose and fatty acids to ATP production in hearts reperfused followiidation rates during reperfusion are low under these conditions, which can result in a decreased recovery of mechanical function. Stimulation of glucose oxidation with the carnitine palmitoyl transferase I inhibitor, Etomoxir, or by directly stimulating pyruvate dehydrogenase activity with dichloroa

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Carnitine requirement of vascular endothelial and smooth muscle cells in imminent ischemia,re vulnerable to oxidative stress than muscle cells and are prone to loose carnitine early during hypoperfusion. This has been suggested by two observations. The first is that incubation of isolated endothelial cells in a low carnitine medium leads to oleate oxidation, dependent upon carnitine addit

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John S. Charnock,Peter L. McLennan,Mahinda Y Abeywardena

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Brian Rodrigues,Michael R. Spooner,David L. Severson

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