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Titlebook: Lipid Mediators in the Immunology of Shock; M. Paubert-Braquet,P. Braquet,M. Foegh Conference proceedings 1987 Plenum Press, New York 1987

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发表于 2025-3-21 18:47:45 | 显示全部楼层 |阅读模式
书目名称Lipid Mediators in the Immunology of Shock
编辑M. Paubert-Braquet,P. Braquet,M. Foegh
视频video
丛书名称NATO Science Series A:
图书封面Titlebook: Lipid Mediators in the Immunology of Shock;  M. Paubert-Braquet,P. Braquet,M. Foegh Conference proceedings 1987 Plenum Press, New York 1987
出版日期Conference proceedings 1987
关键词Antigen; Interferon; Sepsis; Surgery; Trauma; pathophysiology; physiology
版次1
doihttps://doi.org/10.1007/978-1-4613-0919-2
isbn_softcover978-1-4612-8245-7
isbn_ebook978-1-4613-0919-2
copyrightPlenum Press, New York 1987
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发表于 2025-3-21 21:00:18 | 显示全部楼层
The Significance of Pentane Measurements in Mans production. Theoretically, a safe method would determine a lipoperoxide derivating product which is not further catabolized and which arises exclusively from lipoperoxidative pathway. Different procedures were proposed, each one based on the determination of a particular step, during hydroperoxide decomposition. The steps are the following:
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Detection of Pentane as a Measurement of Lipid Peroxidation in Humans Using Gas Chromatography With ketohydroperoxides, and cyclic and bicyclic peroxides (7). These secondary products can in turn decompose to monohydroperoxides, aldehydes such as malonyldialdehyde (MDA), and volatile hydrocarbons (HCs)(7).
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Eicosanoids and The Pathophysiology of Sepsis and Trauma and Subramanian in 1962 (1) that cyclooxygenase inhibitors attenuated the hemodynamic events and prolonged the survival in endotoxemia in dogs has stimulated a substantial interest in the area of the role of eicosanoids in endotoxin shock, bacteremic shock, the sepsis syndrome and septic shock (2–22).
发表于 2025-3-22 10:06:38 | 显示全部楼层
Lipid Metabolism during Starvation and Sepsis in Relation to Fatty Acid Profile in Liver and α-Linolreduced by an inhibitory effect of malonyl-CoA (McGarry et al., 1978). Decreased responses to this inhibition have been reported in starvation (McGarry and Foster, 1979) and by long-chain fatty-acyl-CoA (Mills et al., 1983) in isolated mitochondria. The role of essential fatty acids (EFA) in liver lipids in these conditions is uncertain.
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Role of Free Radical Lipid Peroxidation in Burn and Endotoxin Shock endocrine (Turinsky et al., 1977), neuroendocrine (Cova and Glaviano, 1968; Goodall and Moncrief, 1965), and metabolic (Robinson and Miller, 1981; Wolfe et al., 1977) alterations. Acute endotoxin shock is rapidly produced with . endotoxin (Balis et al., 1978).
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Multiple Organ Failure, Liver Failure and Polyunsaturated Fat Metabolism ineffective, including: ventilation, antibiotics, nutrition, and surgery. The best treatment remains prevention with rapid control of the source and restoration of oxygen transport. To date, no single “magic bullet” has been shown to exist either experimentally or clinically.
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Leukotrienes in Endotoxin Shockukotriene synthesis (10), can be made highly LPS-sensitive by transfer of pure macrophages from LPS-sensitive C3H/HeN mice (11). These data stress the target cell role of macrophages in the in vivo action of LPS (8,11).
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