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Titlebook: Klinische Psychologie; Theoretische und Ide Johannes Helm,Hans-Dieter Rösler,Hans Szewczyk Book 1979 VEB Deutscher Verlag der Wissenschafte

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Marlene Fuchs-Kittowskidouble-strand break repair. In this review, we summarize datafrom biochemical, cell based and in vivo approaches demonstrating that wild-type p53 restrains excessiveand mutagenic DNA exchange events. Thus, p53 appears to exhibit fidelity control of homologous recombination,and recent evidence also s
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Klaus Groscheckption and replication. In 1998, it was found thathtopoI might play another important role in the cellular response to DNA damage -- the so-called htopoIdamage response. Since this initial discovery, many studies have suggested that the htopoI damage responseis involved in DNA repair as well as in ap
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ption and replication. In 1998, it was found thathtopoI might play another important role in the cellular response to DNA damage -- the so-called htopoIdamage response. Since this initial discovery, many studies have suggested that the htopoI damage responseis involved in DNA repair as well as in ap
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Johannes Helmctivity, it has been shown that it can undergostoichiometric (non-enzymatic) interactions with other proteins. This interaction is not only limited tothe tetrameric holoenzyme; the individual subunits of CK2, i.e. the catalytic α-subunit and the regulatoryβ-subunit, can also interact by themselves w
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