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Titlebook: Kosten-Nutzen-Rechnung von Universitäten; Eine Fallstudie am B Heinrich Otruba,Peter Pelizon,Martina Hochreiner Book 1991 Springer-Verlag B

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Heinrich Otruba,Peter Pelizon,Gerhard Manner,Peter Bleier,Martina Hochreinerations on this process led to the conclusion that the organism possesses a special system, the function of which is the removal of the end product of blood coagulation, fibrin, after having fulfilled its physiologic function (. and ., 1889; . and ., 1903; ., 1908; ., 1937). This fibrinolytic system
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Heinrich Otruba,Peter Pelizon,Gerhard Manner,Peter Bleier,Martina Hochreiner either as part of the underlying defect or as a result of liver damage from recurrent episodes of ascending cholangitis. Caroli disease (CD) denotes congenital saccular intrahepatic dilatation of the biliary tree. Caroli syndrome (CS) is a more common disorder in which the bile duct dilatation is a
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Heinrich Otruba,Peter Pelizon,Gerhard Manner,Peter Bleier,Martina Hochreinere various types of fibrocystic diseases, as well as important differential diagnostic entities, can be distinguished from each other confidently using morphologic and functional criteria. Biliary atresia, for example, may not only be suspected by morphologic ultrasound appearance of the liver in com
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Heinrich Otruba,Peter Pelizon,Gerhard Manner,Peter Bleier,Martina Hochreiner liver disease. Caroli’s syndrome (CS) and CD probably represent different presentations of the same continuum. CS refers to CD in association with CHF. CHF/CS and PLD are often part of multisystem disorders associated with fibrocystic renal involvement. These are collectively referred to as “ciliop
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mechanisms of their interaction and cooperative action, in order to provide a basis for the understanding of the effects of fibrinolytic and antifibrinolytic therapeutic agents. Detailed descriptions of the fibrinolytic process and its biochemistry were also given by other authors (., 1956, 1959, 1
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Heinrich Otruba,Peter Pelizon,Gerhard Manner,Peter Bleier,Martina Hochreiner mechanisms of their interaction and cooperative action, in order to provide a basis for the understanding of the effects of fibrinolytic and antifibrinolytic therapeutic agents. Detailed descriptions of the fibrinolytic process and its biochemistry were also given by other authors (., 1956, 1959, 1
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Heinrich Otruba,Peter Pelizon,Gerhard Manner,Peter Bleier,Martina Hochreiner which was described a century ago. First investigations of this process assured that there is in the organism a system capable of removing the fibrin which is formed during blood coagulation after it has fulfilled its physiological function. This fibrinolytic system is specifically adapted to the d
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