Titlebook: JIMD Reports, Volume 19; Johannes Zschocke,Matthias Baumgartner,Verena Pete Book 2015 SSIEM and Springer-Verlag Berlin Heidelberg 2015 Men

Hazardous

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书目名称JIMD Reports, Volume 19影响因子(影响力)学科排名




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书目名称JIMD Reports, Volume 19网络公开度学科排名




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书目名称JIMD Reports, Volume 19被引频次学科排名




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书目名称JIMD Reports, Volume 19年度引用学科排名




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书目名称JIMD Reports, Volume 19读者反馈学科排名

Fecundity

邪恶的你

Yin-Hsiu Chien,Jennifer L. Goldstein,Wuh-Liang Hwu,P. Brian Smith,Ni-Chung Lee,Shu-Chuan Chiang,Advi of 1989. It was the first meeting of its kind and was convened as a forum to review and discuss the phylogeny of some of the cell biological functions that underlie nervous system function, such matters as intercellular communication in diverse, lower organisms, and the electrical excitability of p

溃烂

Mary Ellen Lynch,Nancy L. Potter,Claire D. Coles,Judith L. Fridovich-Keilar space. They are found throughout the animal kingdom in virtually all tissues, including the nervous system where they form electrical synapses. Where gap junctions occur, the adjacent cell membranes are in close apposition, with protein channels spanning a 2–4 nm intercellular space. The junction

Spartan

大方一点

Pankaj Prasun,Sarah Young,Gajja Salomons,Andrea Werneke,Yong-hui Jiang,Eduard Struys,Mikell Paige,Ma complex metabolic disorder in which both genetic and environmental factors play roles in the etiology. Advances in genetic analyses brought tremendous successes in identifying genetic components of common T2D and monogenic form of diabetes such as maturity-onset diabetes of the young (MODY). Large-

处理

放逐某人

Evelina Maines,Grazia Morandi,Francesca Olivieri,Marta Camilot,Paolo Cavarzere,Rossella Gaudino,Franmmon and complex disease, and filaggrin gene (.) was identified as a strong genetic component. Numerous variations of . were identified and European and Asian patients harbor each distinct mutation profiles. Excess non-synonymous variants over synonymous variants of . in general populations indicate

沉积物

A De Novo Variant in Galactose-1-P Uridylyltransferase (,) Leading to Classic Galactosemia,he middle enzyme in the Leloir pathway of galactose metabolism. Patients with CG carry pathogenic loss-of-function mutations in both of their . alleles; the parents of patients are considered obligate carriers. We report here a first exception to that rule – a . . variant in a patient with classic g

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