Titlebook: Ion Channels; Volume 1 Toshio Narahashi Book 1988 Plenum Press, New York 1988 ATP.Calcium.Nucleotide.Peptide.Synapse.biology.cells.kidney.k

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Macromolecular Sites for Specific Neurotoxins and Drugs on Chemosensitive Synapses and Electrical Erts. More recently, cloning studies have been useful in identification of the various subunits of the AChR and presumptive binding sites for the neurotransmitter (Noda ., 1983a; Barnard and Dolly, 1982; Barnard ., 1982). Such studies are of remarkable value in understanding function in nerve and ske

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Intracellular ATP and Cardiac Membrane Currents,% of the control and of ATP to about 90% within 5–10 min when the rate of oxidative phosphorylation was prevented either by replacement of O. with N. or by addition of cyanide (Allen ., 1985). If the supplementary action of the glycolysis was also reduced, the ATP level was reduced more markedly to

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M Currents,sics, UTMB, Galveston, Texas. We were trying to understand the voltage sensitivity of the apparent increase in membrane resistance, and the repetitive firing, produced by muscarine in sympathetic ganglia which Andy Constanti and I had noted in some tests on rat sympathetic neurons (Brown and Constan

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Developmental Changes in Acetylcholine Receptor Channel Properties of Vertebrate Skeletal Muscle,s were greater in denervated muscle than in innervated muscle. This observation was interpreted as an increased duration of the underlying elementary conductance change following denervation. This was later substantiated by noise analysis of ACh-induced membrane current (Neher and Sak-mann, 1976a).

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Intracellular ATP and Cardiac Membrane Currents,for review see Carmeliet, 1978). When cardiac muscle is exposed to anoxic conditions or metabolic inhibitors, or is depleted of substrates, the plateau of the action potential is depressed in the early stage of the metabolic impairment. If the metabolic depression is continued, the electrochemical g

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Calcium Antagonist Receptors,ction of intracellular calcium, and of the many processes that are believed to control the level of cytoplasmic calcium ions, is beyond the scope of this chapter, and the reader is referred to one of the many excellent reviews available on this topic (Rahamimoff ., 1976; Bolton, 1979; Kretsinger, 19