Titlebook: Intracranial Pressure VI; Proceedings of the S J. D. Miller,G. M. Teasdale,A. D. Mendelow Conference proceedings 1986 Springer-Verlag Berli

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RALES

ICP After Experimental Diffuse Head Injuriesration in the monkey (1) and that for equally severe concussions produced by coronal plane angular acceleration, the ICP rises much less dramatically (2). We now present data on more severe diffuse injuries produced by coronal plane acceleration and on concussion produced by horizontal plane acceler

Foam-Cells

Changes in Brain Phosphorus Metabolism in Rats Following Fluid Percussion Injuryent of cerebral circulation and metabolism (Saunders et al. 1979, Lewelt et al. 1980) according to the magnitude of delivered impact pressure. However, study methodologies employed to date generally have been limited to evaluation of changes in brain metabolism at single points in time after fluid p

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Variations in Pressure Volume Index and CSF Outflow Resistance Measured at Different Places in the Cof the risk of herniation is the estimation of the capacity of intracranial compensatory mechanism provided by the pressure volume index (PVI). This is usually determined by bolus injection test into the lateral ventricle. Bolus injection into the lateral ventricle, however, is not always possible i

mechanism

Colonnade

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triptans

Independence of Compliance and CSF Hydrodynamics as an Explanation for Volume Preservation in the Need to describe the two concomitantly, a greater understanding of the interaction between CSF hydrodynamics and neural axis pressure volume relationships has been achieved, but has been limited to acute studies

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Comparison of Pressure Volume Indices Obtained with Constant Rate and Bolus Infusionsrated that Rcsf is determined more accurately by constant rate infusion (Tans, Poortvliet, 1984). For determination of the PVI most investigators use bolus infusions, but constant rate infusions were reported to yield more stable results (Avezaat, Van Eijndhoven 1984). The purpose of the present stu