Titlebook: Interrelationship Among Aging, Cancer and Differentiation; Proceedings of the E Bernard Pullman,Paul O. P. Ts’o,Edmond L. Schneide Conferen

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书目名称Interrelationship Among Aging, Cancer and Differentiation影响因子(影响力)




书目名称Interrelationship Among Aging, Cancer and Differentiation影响因子(影响力)学科排名




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书目名称Interrelationship Among Aging, Cancer and Differentiation网络公开度学科排名




书目名称Interrelationship Among Aging, Cancer and Differentiation被引频次




书目名称Interrelationship Among Aging, Cancer and Differentiation被引频次学科排名




书目名称Interrelationship Among Aging, Cancer and Differentiation年度引用




书目名称Interrelationship Among Aging, Cancer and Differentiation年度引用学科排名




书目名称Interrelationship Among Aging, Cancer and Differentiation读者反馈




书目名称Interrelationship Among Aging, Cancer and Differentiation读者反馈学科排名

安心地散步

Conference proceedings 1985ns of this Symposium was to convene a future meeting to discuss the molecular basis for Interrelationships between aging and cancer when the appropriate scientific knowledge was available. That same year. the 13th Jerusalem Symposium on Quantum Chemistry and Biochemistry entitled ·Carcl­ nogenesls :

一骂死割除

姑姑在炫耀

Transforming Genes of Human Malignancies, tumors has also revealed that the movement of a specific cellular gene, again a homologue of a retroviral transforming gene, to a new regulatory environment alters its expression, thus contributing to tumor development.

产生

不能仁慈

Intermediates in Transcription Initiation and Propagation,essed copy of that gene renders the cell line less oncogenetic, probably by virtue of the body’s ability to mount an immunological defense. Altered gene expression seems likely to be important in the aging process as well, although genes specifically related to normal aging seem not yet to have been found.

故意

Regulators of Growth, Differentiation and the Reversal of Malignancy,genes) there are other genes (soncogenes) that can suppress the action of oncogenes, that reversion does not have to restore all the normal controls, and that the stopping of cell multiplication by inducing differentiation to mature cells can by-pass the genetic abnormalities that give rise to malignancy.

最后一个

Macromolecular Correlates of Cellular Senescence and Cancer,The consequence of both processes is increasing heterogeneity of the cell population, in its DNA structure and potential for gene expression. Such heterogeneity may underlie the age-dependent incidence of clonal-origin diseases such as cancer.

愤怒历史

Deceleration of Cancer Mortality Rates with Age and Time,between cancer and aging, or a simple association between cancer and cumulative exogenous or endogenous factors. Cancer deaths can be expected to represent an ever decreasing proportion of total mortality although a slight rise in absolute numbers should persist.

勋章

Clinical Aspects of Cancer in the Elderly,ness. Patient management must include assessement of general functional and physiologic status and expected survival rates. Surgery, radiation therapy and chemotherapy can be utilized effectively, but individualization of treatment may be necessary. Psychosocial support and measures to maximize quality as well as quantity of life are important.