Titlebook: International Yearbook of Nephrology 1991; Vittorio E. Andreucci,Leon G. Fine Book 1990 Springer Science+Business Media Dordrecht 1990 AID

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Albumin Metabolism in the Nephrotic Syndrome. Implications for Patient Managementminemia in the nephrotic syndrome (7, 10-12). Increased dietary protein intake fails to increase either serum albumin concentration or body albumin pools in patients with the nephrotic syndrome (7, 11-13), or in animals with experimentally induced renal disease (14). Since the human liver normally s

争议的苹果

Risk Factors for Progression of Renal Insufficiency in Diabetic Nephropathy: Therapeutic Implicationrogression of renal insufficiency in diabetic nephropathy is certainly multifactorial (7–10). Only the most studied risk factors will be discussed in the following. Each such factor should ideally be dealt with separately. However, their effects are often superimposed in the studies as in life.

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Endothelin and Endothelium-Derived Relaxing Factor in the Control of Glomerular Filtration and RenalSince the discovery of endothelial-derived relaxing factor (EDRF) (1), a variety of biochemical and mechanical stimuli have been shown to induce the release of endothlial-derived relaxing or contracting factors. It seems likely that EDRF is nitric oxide (2) and it now appears that endothelial-derive

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Role of Glomerular Growth Promoters in Progression of Renal Disease.haracteristic end stage histological pattern ultimately shared by the various diseases has lead to the postulate of existence of common secondary destructive processes triggered by many different primary pathogenic insults (Figure 1). Initial pathogenic insults are presumed to lead to a loss of func

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Plasma Exchange for Renal Diseaseve change in prognosis. The stimulus for new therapeutic initiatives came with the realisation that autoimmune processes were closely associated with events leading to glomerular crescent formation, the histological hallmark of rapidly progressive glomerulonephritis (RPGN). Thus, despite the fact th

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